• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

猫丙烯酰胺神经病中的无髓神经纤维

Unmyelinated nerve fibres in feline acrylamide neuropathy.

作者信息

Post E J

出版信息

Acta Neuropathol. 1978 Apr 26;42(1):19-24. doi: 10.1007/BF01273262.

DOI:10.1007/BF01273262
PMID:207070
Abstract

Electronmicroscope studies have been performed on the greater splanchnic nerve and the nerve to the medial head of gastrocnemius muscle of control and acrylamide poisoned cats. Degeneration of unmyelinated as well as of myelinated fibres was observed in both nerves. In cats severely poisoned with acrylamide, some very large unmyelinated axons undergoing early degeneration were seen in the splanchnic nerve. In the nerve to medial head of gastrocnemius, there was a decrease in the proportion of large diameter unmyelinated axons.

摘要

已对对照猫和丙烯酰胺中毒猫的内脏大神经及腓肠肌内侧头肌支进行了电子显微镜研究。在这两条神经中均观察到了无髓纤维和有髓纤维的变性。在严重丙烯酰胺中毒的猫中,在内脏大神经中可见一些非常大的无髓轴突正在早期变性。在腓肠肌内侧头肌支中,大直径无髓轴突的比例有所下降。

相似文献

1
Unmyelinated nerve fibres in feline acrylamide neuropathy.猫丙烯酰胺神经病中的无髓神经纤维
Acta Neuropathol. 1978 Apr 26;42(1):19-24. doi: 10.1007/BF01273262.
2
Acrylamide autonomic neuropathy in the cat. Part 1. Neurophysiological and histological studies.猫的丙烯酰胺自主神经病变。第1部分。神经生理学和组织学研究。
J Neurol Sci. 1977 Sep;33(3):353-74. doi: 10.1016/0022-510x(77)90133-2.
3
Degeneration of the greater splanchnic nerve by operation.
Anat Anz. 1985;158(3):209-16.
4
Ultrastructural studies of the dying-back process. I. Peripheral nerve terminal and axon degeneration in systemic acrylamide intoxication.轴突回返性病变的超微结构研究。I. 全身性丙烯酰胺中毒时的周围神经终末及轴突变性
J Neuropathol Exp Neurol. 1974 Apr;33(2):260-84. doi: 10.1097/00005072-197404000-00006.
5
Ultrastructural studies of the dying-back process. IV. Differential vulnerability of PNS and CNS fibers in experimental central-peripheral distal axonopathies.轴突逆行性变性过程的超微结构研究。IV. 实验性中枢 - 外周远端轴突病中周围神经系统和中枢神经系统纤维的不同易损性。
J Neuropathol Exp Neurol. 1977 Mar-Apr;36(2):300-20. doi: 10.1097/00005072-197703000-00006.
6
Conduction in unmyelinated fibres in experimental neuropathy.实验性神经病变中无髓鞘纤维的传导
J Neurol Neurosurg Psychiatry. 1972 Apr;35(2):163-9. doi: 10.1136/jnnp.35.2.163.
7
Acrylamide autonomic neuropathy in the cat. Part 2. Effects on mesenteric vascular control.猫的丙烯酰胺自主神经病变。第2部分。对肠系膜血管控制的影响。
J Neurol Sci. 1977 Sep;33(3):375-85. doi: 10.1016/0022-510x(77)90134-4.
8
Nervous system degeneration produced by acrylamide monomer.丙烯酰胺单体导致的神经系统退化。
Environ Health Perspect. 1975 Jun;11:129-33. doi: 10.1289/ehp.7511129.
9
Sequence of motor nerve terminal involvement in acrylamide neuropathy.丙烯酰胺神经病中运动神经终末受累的顺序。
Acta Neuropathol. 1979 Apr 12;46(1-2):57-63. doi: 10.1007/BF00684805.
10
Ultrastructural studies of the dying-back process. III. The evolution of experimental peripheral giant axonal degeneration.轴突逆行性变性过程的超微结构研究。III. 实验性周围性巨大轴突变性的演变
J Neuropathol Exp Neurol. 1977 Mar-Apr;36(2):276-99. doi: 10.1097/00005072-197703000-00005.

引用本文的文献

1
Neurofilaments are nonessential to the pathogenesis of toxicant-induced axonal degeneration.神经丝对毒物诱导的轴突变性的发病机制并非必不可少。
J Neurosci. 2001 Apr 1;21(7):2278-87. doi: 10.1523/JNEUROSCI.21-07-02278.2001.
2
The pathology of allyl chloride neurotoxicity in mice.小鼠中氯丙烯神经毒性的病理学
Acta Neuropathol. 1981;55(2):125-33. doi: 10.1007/BF00699237.

本文引用的文献

1
The normal sural nerve in man. II. Changes in the axons and Schwann cells due to ageing.人类正常的腓肠神经。II. 衰老导致的轴突和施万细胞的变化。
Acta Neuropathol. 1969;13(3):217-39. doi: 10.1007/BF00690643.
2
The normal sural nerve in man. I. Ultrastructure and numbers of fibres and cells.人类正常的腓肠神经。I. 纤维和细胞的超微结构及数量
Acta Neuropathol. 1969;13(3):197-216. doi: 10.1007/BF00690642.
3
The effect of acrylamide on the peripheral nervous system of the baboon.丙烯酰胺对狒狒外周神经系统的影响。
J Neurol Neurosurg Psychiatry. 1970 Dec;33(6):805-16. doi: 10.1136/jnnp.33.6.805.
4
The pathogenesis of dying-back polyneuropathies. I. An ultrastructural study of experimental tri-ortho-cresyl phosphate intoxication in the cat.
J Neuropathol Exp Neurol. 1969 Oct;28(4):571-97. doi: 10.1097/00005072-196910000-00003.
5
The ultrastructure of tri-ortho-cresyl phosphate-poisoning. I. Studies on myelin and axonal alterations in the sciatic nerve.磷酸三邻甲苯酯中毒的超微结构。I. 坐骨神经髓鞘和轴突改变的研究。
Acta Neuropathol. 1967 Oct 20;9(2):159-74. doi: 10.1007/BF00691441.
6
Electron microscopic observations on degeneration and regeneration of unmyelinated fibres.
Brain. 1972;95(2):233-4. doi: 10.1093/brain/95.2.223.
7
Reactions of unmyelinated nerve fibers to injury. An ultrastructural study.无髓神经纤维对损伤的反应。一项超微结构研究。
Brain Res. 1972 Jul 20;42(2):297-309. doi: 10.1016/0006-8993(72)90532-x.
8
The degeneration of unmyelinated axons following nerve section: an ultrastructural study.神经切断后无髓鞘轴突的退变:一项超微结构研究。
J Neurocytol. 1974 Oct;3(4):497-512. doi: 10.1007/BF01098736.
9
A review of acrylamide neurotoxicity. Part I. Properties, uses and human exposure.丙烯酰胺神经毒性综述。第一部分。性质、用途及人体暴露情况。
Can J Neurol Sci. 1974 May;1(2):143-50. doi: 10.1017/s0317167100019739.
10
Motor end-plate fine structure in acrylamide dying-back neuropathy: a sequential morphometric study.
Neurology. 1974 Sep;24(9):849-56. doi: 10.1212/wnl.24.9.849.