Carnegie Mellon University, Pittsburgh, USA.
Cogn Neuropsychol. 2001 Feb 1;18(1):71-92. doi: 10.1080/02643290125986.
Two hypotheses have been advanced concerning the basis of acquired phonological dyslexia. According to the dual-route model, the pattern derives from impaired grapheme-phoneme conversion. According to the phonological impairment hypothesis, it derives from impaired representation and use of phonology. Effects of graphemic complexity and visual similarity observed in studies by Howard and Best (1996), orthographic effects on phoneme counting (Berndt, Haendiges, Mitchum, & Wayland, 1996), and data from patient LB (Derouesne & Beauvois, 1985) have been taken as evidence for an orthographic impairment in phonological dyslexia and therefore against the impaired phonology hypothesis (Coltheart, 1996). We present a computational simulation, results of two behavioral studies, and a critical analysis of the MJ and LB data, which suggest that the "orthographic" deficits in such patients arise from phonological impairments that interact with orthographic properties of stimuli.
关于获得性语音性失读症的基础，有两种假说。根据双重通路模型，这种模式源于字形-音位转换受损。根据语音障碍假说，它源于语音的表示和使用受损。霍华德和贝斯特（1996 年）的研究中观察到的字母复杂性和视觉相似性的影响，拼写法对音位计数的影响（Berndt、Haendiges、Mitchum 和 Wayland，1996 年），以及来自患者 LB 的数据（Derouesne 和 Beauvois，1985 年）被认为是语音性失读症存在拼写法障碍的证据，因此反对语音障碍假说（Coltheart，1996 年）。我们提出了一个计算模拟，两项行为研究的结果，以及对 MJ 和 LB 数据的批判性分析，这些表明此类患者的“拼写法”缺陷源于与刺激的拼写法特性相互作用的语音障碍。
