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字母位置失读症。

Letter position dyslexia.

出版信息

Cogn Neuropsychol. 2001 Dec 1;18(8):673-96. doi: 10.1080/02643290143000051.

DOI:10.1080/02643290143000051
PMID:20945233
Abstract

Many word-reading models assume that the early stages of reading involve a separate process of letter position encoding. However, neuropsychological evidence for the existence and selectivity of this function has been rather indirect, coming mainly from position preservation in migrations between words in attentional dyslexia, and from nonselective reading deficits. No pure demonstration of selective impairment of letter position function has yet been made. In this paper two Hebrew-speaking acquired dyslexic patients with occipito-parietal lesions are presented who suffer from a highly selective deficit to letter position encoding. As a result of this deficit, they predominantly make errors of letter migration within words (such as reading "broad" for "board") in a wide variety of tasks: oral reading, lexical decision, same-different decision, and letter location. The deficit is specific to orthographic material, and is manifested mainly in medial letter positions. The implications of the findings to models of reading and attention are discussed.

摘要

许多单词阅读模型假设阅读的早期阶段涉及到一个单独的字母位置编码过程。然而,神经心理学证据表明这种功能的存在和选择性是相当间接的,主要来自于注意力障碍性阅读中单词之间迁移时的位置保持,以及非选择性阅读缺陷。目前还没有对字母位置功能选择性损伤的纯证明。本文介绍了两位患有顶枕叶病变的希伯来语后天性阅读障碍患者,他们存在高度选择性的字母位置编码缺陷。由于这种缺陷,他们在各种任务中主要出现字母在单词内迁移的错误(例如将“broad”读成“board”):口头阅读、词汇判断、相同/不同判断和字母位置。这种缺陷是针对拼字材料的,主要表现在中间字母位置。研究结果对阅读和注意模型的影响进行了讨论。

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