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Impaired mTORC2 signaling in catecholaminergic neurons exaggerates high fat diet-induced hyperphagia.儿茶酚胺能神经元中 mTORC2 信号的损伤会加剧高脂肪饮食诱导的过度摄食。
Heliyon. 2015 Sep 21;1(1):e00025. doi: 10.1016/j.heliyon.2015.e00025. eCollection 2015 Sep.
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Comprehensive Map of Molecules Implicated in Obesity.肥胖相关分子综合图谱
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Moving beyond energy homeostasis: new roles for glucagon-like peptide-1 in food and drug reward.超越能量稳态：胰高血糖素样肽-1在食物和药物奖赏中的新作用
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Neuronal ablation of p-Akt at Ser473 leads to altered 5-HT1A/2A receptor function.在丝氨酸473位点对p-Akt进行神经元消融会导致5-羟色胺1A/2A受体功能改变。
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本文引用的文献

1
Insulin reveals Akt signaling as a novel regulator of norepinephrine transporter trafficking and norepinephrine homeostasis.胰岛素揭示 Akt 信号作为去甲肾上腺素转运体运输和去甲肾上腺素动态平衡的新型调节因子。
J Neurosci. 2010 Aug 25;30(34):11305-16. doi: 10.1523/JNEUROSCI.0126-10.2010.
2
Hypothalamic proinflammatory lipid accumulation, inflammation, and insulin resistance in rats fed a high-fat diet.高脂饮食喂养大鼠下丘脑促炎脂质蓄积、炎症及胰岛素抵抗
Am J Physiol Endocrinol Metab. 2009 May;296(5):E1003-12. doi: 10.1152/ajpendo.90377.2008. Epub 2008 Dec 30.
3
Insulin, leptin, and food reward: update 2008.胰岛素、瘦素与食物奖赏：2008年更新
Am J Physiol Regul Integr Comp Physiol. 2009 Jan;296(1):R9-R19. doi: 10.1152/ajpregu.90725.2008. Epub 2008 Oct 22.
4
Hypoinsulinemia regulates amphetamine-induced reverse transport of dopamine.低胰岛素血症调节苯丙胺诱导的多巴胺逆向转运。
PLoS Biol. 2007 Oct 16;5(10):e274. doi: 10.1371/journal.pbio.0050274.
5
Is dopamine a physiologically relevant mediator of feeding behavior?多巴胺是进食行为的生理相关介质吗？
Trends Neurosci. 2007 Aug;30(8):375-81. doi: 10.1016/j.tins.2007.06.004. Epub 2007 Jun 29.
6
Brain dopamine and obesity.脑多巴胺与肥胖
Lancet. 2001 Feb 3;357(9253):354-7. doi: 10.1016/s0140-6736(00)03643-6.

Insulin regulation of monoamine signaling: pathway to obesity.

作者信息

Niswender Kevin D, Daws Lynette C, Avison Malcolm J, Galli Aurelio

机构信息

Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.

出版信息

Neuropsychopharmacology. 2011 Jan;36(1):359-60. doi: 10.1038/npp.2010.167.

DOI:10.1038/npp.2010.167
PMID:21116253
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3055525/
Abstract
摘要