Wang J
Wuxi Fifth People's Hospital.
Zhongguo Yi Xue Ke Xue Yuan Xue Bao. 1990 Feb;12(1):61-7.
Based on the analysis of heritable autoregulatory functions and adaptive developments which occur long course of in response to inner and outer environment, FCMCK therapy was first designed to mobilize the autoregulatory system and resist the stress of acute hemorrhagic cerebral stroke. In this paper, 44 cases of acute hemorrhagic cerebral stroke were treated with FCMCK therapy, with another 44 cases treated with mannitol as control. The result showed that the mortality rate of the treated group was 4.5% (2/44), significantly lower than that of the control (47.7%, 21/44, P less than 0.01). FCMCK therapy in acute hemorrhagic cerebral stroke has the following advantages: 1) effectiveness in maintenance of adequate blood pressure; 2) effectiveness in reduction of cardiac arrhythmias and other complications; 3) i.v. drip of Ca and repeated use of digitalis shows atoxic effect; and 4) respiratory failure improves without stopping i.v. drip of Mg. The mechanism of FCMCK therapy is briefly discussed by the authors.
基于对在长期响应内外部环境过程中发生的遗传性自身调节功能和适应性发展的分析，FCMCK疗法最初被设计用于调动自身调节系统并抵抗急性出血性脑卒中的应激。本文中，44例急性出血性脑卒中患者接受了FCMCK疗法治疗，另外44例患者用甘露醇治疗作为对照。结果显示，治疗组的死亡率为4.5%（2/44），显著低于对照组（47.7%，21/44，P<0.01）。FCMCK疗法治疗急性出血性脑卒中有以下优点：1）在维持足够血压方面有效；2）在减少心律失常和其他并发症方面有效；3）静脉滴注钙和重复使用洋地黄显示无毒性作用；4）呼吸衰竭在不停用静脉滴注镁的情况下得到改善。作者简要讨论了FCMCK疗法的机制。
