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单核细胞增生李斯特菌向CD8 + T细胞的呈递需要溶血素的分泌和细胞内细菌生长。

Presentation of Listeria monocytogenes to CD8+ T cells requires secretion of hemolysin and intracellular bacterial growth.

作者信息

Brunt L M, Portnoy D A, Unanue E R

机构信息

Department of Pathology, Washington University School of Medicine, St. Louis, MO 63110.

出版信息

J Immunol. 1990 Dec 1;145(11):3540-6.

PMID:2147195
Abstract

Cell-mediated immunity to Listeria monocytogenes (LM) involves both CD4+ and CD8+ T cell responses. An important virulence factor in the pathogenesis of infection and development of protective immunity to LM is secretion of the sulfhydryl-activated hemolysin (Hly), listeriolysin. Listeria secretion of Hly allows LM to escape the endosomal compartment and enter the cytosol of the cell where intracellular growth can occur. We developed a system using bone marrow macrophages cultured in CSF-1 or IFN-gamma for comparing the response of CD4+ or CD8+ T cells to heat-killed, live Hly-, and live Hly+ LM. Macrophages grown in CSF were permissive for intracellular growth of Hly+ but not Hly- LM. CD8+ T cells recognized Hly+ LM but not HK or Hly- LM pulsed macrophages. However, CD4+ T cells recognized all three Listeria preparations fed to IFN-gamma-treated macrophages. These results suggest that both heat-killed LM and live LM efficiently enter the exogenous pathway for class II Ag processing and presentation. In contrast, only Hly+ LM activates the class I pathway, probably as a result of Hly+ bacterial replication within the cytosolic compartment.

摘要

针对单核细胞增生李斯特菌(LM)的细胞介导免疫涉及CD4⁺和CD8⁺ T细胞应答。硫氢基激活溶血素(Hly)即李斯特菌溶血素的分泌是LM感染发病机制及保护性免疫形成过程中的一个重要毒力因子。Hly的分泌使LM能够逃离内体区室并进入细胞胞质溶胶,进而在胞内生长。我们开发了一个系统,使用在集落刺激因子-1(CSF-1)或γ干扰素中培养的骨髓巨噬细胞,来比较CD4⁺或CD8⁺ T细胞对热灭活、活的Hly⁻和活的Hly⁺ LM的应答。在CSF中生长的巨噬细胞允许Hly⁺ LM而非Hly⁻ LM在胞内生长。CD8⁺ T细胞识别Hly⁺ LM,但不识别热灭活或Hly⁻ LM刺激的巨噬细胞。然而,CD4⁺ T细胞识别所有三种喂食给γ干扰素处理的巨噬细胞的李斯特菌制剂。这些结果表明,热灭活的LM和活的LM都能有效地进入Ⅱ类抗原加工和呈递的外源性途径。相比之下,只有Hly⁺ LM激活Ⅰ类途径,这可能是由于Hly⁺细菌在胞质区室中复制的结果。

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