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A role for the cancer-associated miR-106b~25 cluster in neuronal stem cells.

作者信息

Peck Barrie, Schulze Almut

出版信息

Aging (Albany NY). 2011 Apr;3(4):329-31. doi: 10.18632/aging.100302.

DOI:10.18632/aging.100302
PMID:21483041
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3117442/
Abstract
摘要

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本文引用的文献

1
The microRNA cluster miR-106b~25 regulates adult neural stem/progenitor cell proliferation and neuronal differentiation.微小RNA簇miR-106b~25调控成年神经干细胞/祖细胞的增殖和神经元分化。
Aging (Albany NY). 2011 Feb;3(2):108-24. doi: 10.18632/aging.100285.
2
FoxOs enforce a progression checkpoint to constrain mTORC1-activated renal tumorigenesis.FoxOs 建立一个进展检查点来限制 mTORC1 激活的肾肿瘤发生。
Cancer Cell. 2010 Nov 16;18(5):472-84. doi: 10.1016/j.ccr.2010.10.019.
3
Identification of the miR-106b~25 microRNA cluster as a proto-oncogenic PTEN-targeting intron that cooperates with its host gene MCM7 in transformation.鉴定 miR-106b~25 微 RNA 簇为原癌基因 PTEN 靶向内含子，与宿主基因 MCM7 合作参与转化。
Sci Signal. 2010 Apr 13;3(117):ra29. doi: 10.1126/scisignal.2000594.
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Foxo3a regulates apoptosis by negatively targeting miR-21.Foxo3a 通过负向靶向 miR-21 调节细胞凋亡。
J Biol Chem. 2010 May 28;285(22):16958-66. doi: 10.1074/jbc.M109.093005. Epub 2010 Apr 6.
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MicroRNA-155 regulates cell survival, growth, and chemosensitivity by targeting FOXO3a in breast cancer.MicroRNA-155 通过靶向乳腺癌中的 FOXO3a 调节细胞存活、生长和化疗敏感性。
J Biol Chem. 2010 Jun 4;285(23):17869-79. doi: 10.1074/jbc.M110.101055. Epub 2010 Apr 6.
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Roles of small regulatory RNAs in determining neuronal identity.小调控 RNA 在决定神经元身份中的作用。
Nat Rev Neurosci. 2010 May;11(5):329-38. doi: 10.1038/nrn2739. Epub 2010 Mar 31.
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Neural mechanisms of ageing and cognitive decline.衰老和认知能力下降的神经机制。
Nature. 2010 Mar 25;464(7288):529-35. doi: 10.1038/nature08983.
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FoxOs cooperatively regulate diverse pathways governing neural stem cell homeostasis.FoxOs 协同调控多种通路，调节神经干细胞的稳态。
Cell Stem Cell. 2009 Nov 6;5(5):540-53. doi: 10.1016/j.stem.2009.09.013.
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FoxO3 regulates neural stem cell homeostasis.FoxO3 调节神经干细胞稳态。
Cell Stem Cell. 2009 Nov 6;5(5):527-39. doi: 10.1016/j.stem.2009.09.014.
10
The miR-106b-25 polycistron, activated by genomic amplification, functions as an oncogene by suppressing p21 and Bim.由基因组扩增激活的miR-106b-25多顺反子通过抑制p21和Bim发挥癌基因的作用。
Gastroenterology. 2009 May;136(5):1689-700. doi: 10.1053/j.gastro.2009.02.002.