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蛋白聚糖在导致胎盘血栓形成和胎儿生长受限中的作用。

The role of proteoglycans in contributing to placental thrombosis and fetal growth restriction.

作者信息

Said Joanne M

机构信息

Level 7 Research Precinct, The Royal Women's Hospital, Locked Bag 300, Parkville, VIC 3052, Australia; Department of Obstetrics and Gynaecology, University of Melbourne, Parkville 3052, Australia.

出版信息

J Pregnancy. 2011;2011:928381. doi: 10.1155/2011/928381. Epub 2011 Mar 3.

Abstract

Fetal growth restriction is an important pregnancy complication that has major consequences for the fetus and neonate as well as an increased risk of long-term morbidity extending into adulthood. The precise aetiology of most cases of fetal growth restriction is unknown although placental thrombosis is a common feature in many of these cases. This paper will outline the potential role of proteoglycans in contributing to placental thrombosis and fetal growth restriction.

摘要

胎儿生长受限是一种重要的妊娠并发症,对胎儿和新生儿有重大影响,并且成年后患长期疾病的风险也会增加。尽管胎盘血栓形成是许多胎儿生长受限病例的常见特征,但大多数病例的确切病因尚不清楚。本文将概述蛋白聚糖在导致胎盘血栓形成和胎儿生长受限方面的潜在作用。

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引用本文的文献

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Evaluating the Effect of Utrogestan on Idiopathic Intrauterine Growth Retardation.
Adv Biomed Res. 2020 Sep 30;9:45. doi: 10.4103/abr.abr_27_20. eCollection 2020.

本文引用的文献

1
Placental biglycan expression is decreased in human idiopathic fetal growth restriction.
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2
Decorin expression is decreased in human idiopathic fetal growth restriction.
Reprod Fertil Dev. 2010;22(6):949-55. doi: 10.1071/RD09240.
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