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Mitochondrial dysfunction during sepsis: still more questions than answers.

作者信息

Exline Matthew C, Crouser Elliott D

出版信息

Crit Care Med. 2011 May;39(5):1216-7. doi: 10.1097/CCM.0b013e31821487cb.

DOI:10.1097/CCM.0b013e31821487cb
PMID:21603080
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3097475/
Abstract
摘要

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本文引用的文献

1
Bioenergetic failure of human peripheral blood monocytes in patients with septic shock is mediated by reduced F1Fo adenosine-5'-triphosphate synthase activity.败血症性休克患者外周血单核细胞的生物能量衰竭是由 F1Fo 腺苷-5'-三磷酸合酶活性降低介导的。
Crit Care Med. 2011 May;39(5):1056-63. doi: 10.1097/CCM.0b013e31820eda5c.
2
Temporal increase of platelet mitochondrial respiration is negatively associated with clinical outcome in patients with sepsis.血小板线粒体呼吸的时间增加与脓毒症患者的临床结局呈负相关。
Crit Care. 2010;14(6):R214. doi: 10.1186/cc9337. Epub 2010 Nov 24.
3
Survival in critical illness is associated with early activation of mitochondrial biogenesis.危重病患者的存活率与线粒体生物发生的早期激活有关。
Am J Respir Crit Care Med. 2010 Sep 15;182(6):745-51. doi: 10.1164/rccm.201003-0326OC. Epub 2010 Jun 10.
4
Bundled care for septic shock: an analysis of clinical trials.脓毒性休克的捆绑式护理：临床试验分析。
Crit Care Med. 2010 Feb;38(2):668-78. doi: 10.1097/CCM.0b013e3181cb0ddf.
5
Antioxidant treatment reverses mitochondrial dysfunction in a sepsis animal model.抗氧化剂治疗可逆转脓毒症动物模型中的线粒体功能障碍。
Mitochondrion. 2008 Jun;8(3):211-8. doi: 10.1016/j.mito.2008.03.002. Epub 2008 Mar 10.
6
Oxygen consumption of human peripheral blood mononuclear cells in severe human sepsis.严重人类脓毒症中人类外周血单个核细胞的氧消耗
Crit Care Med. 2007 Dec;35(12):2702-8. doi: 10.1097/01.ccm.0000295593.25106.c4.
7
Suppression of mitochondrial ATPase inhibitor protein (IF1) in the liver of late septic rats.
Biochim Biophys Acta. 2007 Jul;1767(7):888-96. doi: 10.1016/j.bbabio.2007.03.009. Epub 2007 Apr 3.
8
A network-based analysis of systemic inflammation in humans.基于网络的人类全身炎症分析。
Nature. 2005 Oct 13;437(7061):1032-7. doi: 10.1038/nature03985. Epub 2005 Aug 31.
9
Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome.脓毒症休克和多器官功能障碍综合征中的线粒体功能障碍
Mitochondrion. 2004 Sep;4(5-6):729-41. doi: 10.1016/j.mito.2004.07.023. Epub 2004 Oct 2.
10
Multiorgan failure is an adaptive, endocrine-mediated, metabolic response to overwhelming systemic inflammation.多器官功能衰竭是对严重全身性炎症的一种适应性、内分泌介导的代谢反应。
Lancet. 2004;364(9433):545-8. doi: 10.1016/S0140-6736(04)16815-3.