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应激过程中脑和心肌中生长激素、环核苷酸水平的变化以及蛋白质代谢状态，以及应激后心肌坏死情况

[Changes in the levels of somatotropin and cyclic nucleotides and the state of protein metabolism in the brain and myocardium during stress as well as in myocardial necrosis following stress].

作者信息

Iakushev V S, Kuripka V I, Belokon' L E, Mironova E V, Krisanova N V, Davydov V V, Skurygin V P

出版信息

Vopr Med Khim. 1990 Jan-Feb;36(1):19-23.

PMID:2160753
Abstract

Stress itself as well as the subsequent myocardium necrosis developed after stress were mainly responsible for distinct decrease in concentration of somatotropin, somatomedin and for impairments in protein synthesis accompanied by decrease in proteins half-life in brain hemispheres and in non-ischemized heart left ventricle.

摘要

应激本身以及应激后发生的心肌坏死是导致生长激素、生长调节素浓度明显降低以及蛋白质合成受损的主要原因，蛋白质合成受损伴随着大脑半球和未缺血的心脏左心室中蛋白质半衰期的缩短。

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1
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