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Interactions of murine leukemia virus (MuLV) with isolated lymphocytes. IV. The role of mitogen-induced cellular DNA synthesis in virus infection and replication.

作者信息

Cerny J, Isaak D D

出版信息

Int J Cancer. 1979 Feb;23(2):260-8. doi: 10.1002/ijc.2910230218.

DOI:10.1002/ijc.2910230218
PMID:216643
Abstract
摘要

相似文献

1
Interactions of murine leukemia virus (MuLV) with isolated lymphocytes. IV. The role of mitogen-induced cellular DNA synthesis in virus infection and replication.
Int J Cancer. 1979 Feb;23(2):260-8. doi: 10.1002/ijc.2910230218.
2
Interactions of murine leukemia virus (MuLV) with isolated lymphocytes. II. Infections of B and T cells with Friend virus complex indiffusion chambers and in vitro: effect of polyclonal mitogens.鼠白血病病毒(MuLV)与分离淋巴细胞的相互作用。II. 用弗瑞德病毒复合物在扩散盒中和体外感染B细胞和T细胞:多克隆有丝分裂原的作用
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Interactions of murine leukemia virus (MuLV) with isolated lymphocytes. I. Virus replication in lymphocytes infected with Friend virus and cultures in diffusion chambers in vivo.鼠白血病病毒(MuLV)与分离淋巴细胞的相互作用。I. 病毒在感染弗氏病毒的淋巴细胞中的复制以及体内扩散小室中的培养。
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The effect of glutamine on murine splenic leukocyte responses to T and B cell mitogens.谷氨酰胺对小鼠脾脏白细胞对T细胞和B细胞有丝分裂原反应的影响。
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In vitro generation of tumor-specific cytotoxic lymphocytes. Secondary allogeneic mixed tumor lymphocyte culture of normal murine spleen cells.肿瘤特异性细胞毒性淋巴细胞的体外生成。正常小鼠脾细胞的二次同种异体混合肿瘤淋巴细胞培养。
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Endogenous retroviral env expression in primary murine leukemias: lack of xenotropic antigens but presence of distinct mink cell focus-forming env subtypes correlating with ecotropic virus inoculated and mouse strain.内源性逆转录病毒env在原发性小鼠白血病中的表达:缺乏嗜异性抗原,但存在与嗜亲性病毒接种及小鼠品系相关的不同的貂细胞灶形成env亚型。
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Induction of endogenous murine C-type virus in spleen cell cultures treated with mitogens and 5-bromo-2'-deoxyuridine.用促细胞分裂剂和5-溴-2'-脱氧尿苷处理的脾细胞培养物中内源性鼠C型病毒的诱导。
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Friend murine leukemia virus-induced leukemia is associated with the formation of mink cell focus-inducing viruses and is blocked in mice expressing endogenous mink cell focus-inducing xenotropic viral envelope genes.Friend小鼠白血病病毒诱导的白血病与貂细胞集落诱导病毒的形成有关,并且在表达内源性貂细胞集落诱导异种嗜性病毒包膜基因的小鼠中受到阻断。
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