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针刺镇痛的神经生理学基础。

Neurophysiological basis of acupuncture analgesia.

作者信息

Hsiang-Tung C

出版信息

Sci Sin. 1978 Nov-Dec;21(6):829-46.

PMID:217085
Abstract

This article presents a view about the neural mechanism of acupuncture analgesia and the clinical and experimental evidences on which this view is based. The analgesic effect of acupuncture is considered essentially a manifestation of the interaction of afferent impulses from the region of pain and those from the point of acupuncture. It begins with a general information leading to the development of this sensory interaction theory in interpretation of acupuncture analgesia. The nature of the acupuncture feeling, the receptors activated by acupuncture, the types of fibers involved in transmission of the afferent impulses produced by acupuncture and the processes of interaction taking place at different levels of the central nervous system are described with particular reference to the integrative action of extralemniscal system including the bulbar reticular formation and the intralaminar structures of thalamus. The possible rôle played by the putative synaptic neurotransmitters such as serotonin is only very briefly mentioned.

摘要

本文提出了一种关于针刺镇痛神经机制的观点以及该观点所基于的临床和实验证据。针刺的镇痛作用本质上被认为是来自疼痛区域的传入冲动与来自针刺穴位的传入冲动相互作用的表现。文章首先介绍了导致这种感觉相互作用理论发展以解释针刺镇痛的一般信息。特别参考了包括延髓网状结构和丘脑板内核结构在内的脊髓外系统的整合作用,描述了针刺感觉的性质、针刺激活的感受器、针刺产生的传入冲动传导所涉及的纤维类型以及在中枢神经系统不同水平发生的相互作用过程。对诸如血清素等假定的突触神经递质可能发挥的作用仅作了非常简要的提及。

相似文献

1
Neurophysiological basis of acupuncture analgesia.针刺镇痛的神经生理学基础。
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Neuronal GRK2 regulates microglial activation and contributes to electroacupuncture analgesia on inflammatory pain in mice.神经元 GRK2 调节小胶质细胞激活,并有助于电针对小鼠炎症性疼痛的镇痛作用。
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J Neurol. 1985;232(2):79-82. doi: 10.1007/BF00313905.
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Attenuation of somatosensory evoked potentials by acupuncture and tactile skin stimulation in man.针刺和皮肤触觉刺激对人体体感诱发电位的衰减作用。
Brain Topogr. 1991 Fall;4(1):37-46. doi: 10.1007/BF01129664.