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颞骨的解剖学考虑作为贝尔面瘫的发病机制。

Anatomical consideration of the temporal bone as a pathogenesis of Bell's palsy.

机构信息

Department of Otorhinolaryngology, Inje University College of Medicine, Goyang, Republic of Korea.

出版信息

Med Hypotheses. 2011 Nov;77(5):705-7. doi: 10.1016/j.mehy.2011.07.017. Epub 2011 Aug 23.

Abstract

Much controversy surrounds the etiology and management of Bell's palsy, and the pathogenetic mechanisms underlying Bell's palsy remain obscure despite the extensive body of relevant research. The pathological condition of Bell's palsy is almost an inflammatory reaction compressing the facial nerve in the fallopian canal, particularly in the narrowest labyrinthine segment, followed by demyelinating neural change. As one of the best method for the visualization of the inflamed facial nerve in the intratemporal segment, temporal bone CT enable us not only to measure the exact length and thickness of individual segments of the facial nerve canal but also to view variable anatomic variations in the intratemporal facial canal. Some suggest that anatomical variation may predispose the nerve to inflammatory processes and also there can be peculiar structures of the temporal bone that are vulnerable to inflammation or neural compression injury.

摘要

贝尔氏麻痹的病因和治疗存在很多争议,尽管相关研究很多,但贝尔氏麻痹的发病机制仍不清楚。贝尔氏麻痹的病理状况几乎是一种炎症反应,压迫面神经在咽鼓管,特别是在最狭窄的迷路段,随后是脱髓鞘的神经变化。颞骨 CT 作为显示颞骨内段炎症性面神经的最佳方法之一,不仅使我们能够测量面神经管各段的确切长度和厚度,还可以观察到颞骨内面神经管的各种解剖变异。有人认为,解剖变异可能使神经容易发生炎症过程,颞骨也可能存在易受炎症或神经压迫损伤的特殊结构。

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