Maurer K, Leitner H, Schäfer E, Hopf H C
Dtsch Med Wochenschr. 1979 Apr 13;104(15):546-50. doi: 10.1055/s-0028-1103943.
Within the first 10 ms after a sine-shaped sound wave (tone pip) seven small-amplitude potentials can be recorded in persons with normal hearing and normal brainstem functions. These components, in the nanovolt range, correspond to the electrical activity of various pathways of the auditory tract. In accordance with this view the resulting potentials were assigned to the following structures in the region of the periphery and the brainstem: component I corresponds to the cochlea or acoustic nerve (receptor, II to the cochlear nucleus (medulla), III to the upper olive (caudal pons), IV to the lateral lemniscus (rostral pons), V to the inferior colliculus (midbrain), VI to the medial geniculate body (diencephalon), VII to the acoustic radiation (cortex). Clinically well defined lesions of the acoustic nerve and brainstem indicate that there is a close topographical relationship between the clinical localisation and absence or delay of the individual components.
在正弦形声波(短音)出现后的最初10毫秒内,听力正常且脑干功能正常的人可以记录到七个小振幅电位。这些处于纳伏范围内的成分,对应于听觉传导通路各部分的电活动。根据这一观点,所产生的电位被归属于外周和脑干区域的以下结构:成分I对应于耳蜗或听神经(感受器),II对应于蜗神经核(延髓),III对应于上橄榄核(脑桥尾部),IV对应于外侧丘系(脑桥头部),V对应于下丘(中脑),VI对应于内侧膝状体(间脑),VII对应于听辐射(皮层)。听神经和脑干临床上明确的病变表明,临床定位与各个成分的缺失或延迟之间存在密切的拓扑关系。