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鼠疫耶尔森氏菌外膜蛋白 A(OmpA)促进了在小鼠体内的存活和毒力。

The outer membrane protein A (OmpA) of Yersinia pestis promotes intracellular survival and virulence in mice.

机构信息

Department of Microbiology and Immunology, University of Miami, Miller School of Medicine, 1600 NW 10th Ave, Miami, FL 33136, USA.

出版信息

Microb Pathog. 2012 Jan;52(1):41-6. doi: 10.1016/j.micpath.2011.09.009. Epub 2011 Oct 18.

Abstract

The plague bacterium Yersinia pestis has a number of well-described strategies to protect itself from both host cells and soluble factors. In an effort to identify additional anti-host factors, we employed a transposon site hybridization (TraSH)-based approach to screen 10(5)Y. pestis mutants in an in vitro infection system. In addition to loci encoding various components of the well-characterized type III secretion system (T3SS), our screen unambiguously identified ompA as a pro-survival gene. We go on to show that an engineered Y. pestis ΔompA strain, as well as a ΔompA strain of the closely related pathogen Yersinia pseudotuberculosis, have fully functioning T3SSs but are specifically defective in surviving within macrophages. Additionally, the Y. pestis ΔompA strain was out competed by the wild-type strain in a mouse co-infection assay. Unlike in other bacterial pathogens in which OmpA can promote adherence, invasion, or serum resistance, the OmpA of Y. pestis is restricted to enhancing intracellular survival. Our data show that OmpA of the pathogenic Yersinia is a virulence factor on par with the T3SS.

摘要

鼠疫耶尔森菌有许多描述明确的策略来保护自己免受宿主细胞和可溶性因子的侵害。为了鉴定其他抗宿主因子,我们采用转座子位点杂交(TraSH)为基础的方法,在体外感染系统中筛选 10(5)Y。鼠疫耶尔森氏菌突变体。除了编码特征明确的 III 型分泌系统(T3SS)的各种成分的基因座外,我们的筛选还明确鉴定出 ompA 为一个生存基因。我们接着表明,工程 Y。鼠疫耶尔森氏菌ΔompA 株,以及密切相关的病原体假结核耶尔森氏菌的ΔompA 株,具有完全功能的 T3SS,但在巨噬细胞内生存能力特别差。此外,在小鼠混合感染实验中,Y。鼠疫耶尔森氏菌ΔompA 株的竞争能力低于野生型株。与其他细菌病原体不同,OmpA 可以促进粘附、入侵或血清抵抗,Y。鼠疫耶尔森氏菌的 OmpA 仅限于增强细胞内生存能力。我们的数据表明,致病性耶尔森氏菌的 OmpA 是一种与 T3SS 相当的毒力因子。

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