相似文献
1
Channeling dysglycemia: ion-channel variations perturbing glucose homeostasis.
Trends Endocrinol Metab. 2012 Jan;23(1):41-8. doi: 10.1016/j.tem.2011.09.004. Epub 2011 Nov 29.
2
Decreasing cx36 gap junction coupling compensates for overactive KATP channels to restore insulin secretion and prevent hyperglycemia in a mouse model of neonatal diabetes.
Diabetes. 2014 May;63(5):1685-97. doi: 10.2337/db13-1048. Epub 2014 Jan 23.
3
Functional and Metabolomic Consequences of K Channel Inactivation in Human Islets.
Diabetes. 2017 Jul;66(7):1901-1913. doi: 10.2337/db17-0029. Epub 2017 Apr 25.
4
INGAP-PP up-regulates the expression of genes and proteins related to K+ ATP channels and ameliorates Ca2+ handling in cultured adult rat islets.
Regul Pept. 2008 Jun 5;148(1-3):39-45. doi: 10.1016/j.regpep.2008.02.008. Epub 2008 Mar 4.
5
ATP-sensitive potassium channelopathies: focus on insulin secretion.
J Clin Invest. 2005 Aug;115(8):2047-58. doi: 10.1172/JCI25495.
6
Glucose controls cytosolic Ca2+ and insulin secretion in mouse islets lacking adenosine triphosphate-sensitive K+ channels owing to a knockout of the pore-forming subunit Kir6.2.
Endocrinology. 2009 Jan;150(1):33-45. doi: 10.1210/en.2008-0617. Epub 2008 Sep 11.
7
Acute effect of 3β-hidroxihop-22(29)ene on insulin secretion is mediated by GLP-1, potassium and calcium channels for the glucose homeostasis.
J Steroid Biochem Mol Biol. 2015 Jun;150:112-22. doi: 10.1016/j.jsbmb.2015.03.011. Epub 2015 Apr 2.
8
Critical role of gap junction coupled KATP channel activity for regulated insulin secretion.
PLoS Biol. 2006 Feb;4(2):e26. doi: 10.1371/journal.pbio.0040026. Epub 2006 Jan 17.
9
Insulin secretion from beta cells within intact islets: location matters.
Clin Exp Pharmacol Physiol. 2015 Apr;42(4):406-14. doi: 10.1111/1440-1681.12368.
10
Increased uncoupling protein (UCP) activity in Drosophila insulin-producing neurons attenuates insulin signaling and extends lifespan.
Aging (Albany NY). 2009 Jul 21;1(8):699-713. doi: 10.18632/aging.100067.
引用本文的文献
1
Inwardly rectifying potassium channels: Critical insights for insect species and Apis mellifera.
Channels (Austin). 2025 Dec;19(1):2529250. doi: 10.1080/19336950.2025.2529250. Epub 2025 Jul 10.
2
Congenital Hyperinsulinism Caused by Mutations in Gene Associated with Early-Onset Neonatal Hypoglycemia: Genetic Heterogeneity Correlated with Phenotypic Variability.
Int J Mol Sci. 2024 May 19;25(10):5533. doi: 10.3390/ijms25105533.
3
Potential Applications of Gliclazide in Treating Type 1 Diabetes Mellitus: Formulation with Bile Acids and Probiotics.
Eur J Drug Metab Pharmacokinet. 2018 Jun;43(3):269-280. doi: 10.1007/s13318-017-0441-y.
4
Sulfonylurea challenge test in subjects diagnosed with type 1 diabetes mellitus.
Pediatr Diabetes. 2017 Dec;18(8):777-784. doi: 10.1111/pedi.12489. Epub 2017 Jan 23.
5
ABCC8 Single Nucleotide Polymorphisms are Associated with Cerebral Edema in Severe TBI.
Neurocrit Care. 2017 Apr;26(2):213-224. doi: 10.1007/s12028-016-0309-z.
6
Discovery and characterization of a potent and selective inhibitor of Aedes aegypti inward rectifier potassium channels.
PLoS One. 2014 Nov 6;9(11):e110772. doi: 10.1371/journal.pone.0110772. eCollection 2014.
7
Molecular and functional characterization of Anopheles gambiae inward rectifier potassium (Kir1) channels: a novel role in egg production.
Insect Biochem Mol Biol. 2014 Aug;51:10-9. doi: 10.1016/j.ibmb.2014.05.002. Epub 2014 May 20.
8
Direct activation of β-cell KATP channels with a novel xanthine derivative.
Mol Pharmacol. 2014 Jun;85(6):858-65. doi: 10.1124/mol.114.091884. Epub 2014 Mar 19.
9
Ion channel associated diseases: overview of molecular mechanisms.
Chem Rev. 2012 Dec 12;112(12):6319-33. doi: 10.1021/cr300360k. Epub 2012 Nov 14.
本文引用的文献
1
Diazoxide-unresponsive congenital hyperinsulinism in children with dominant mutations of the β-cell sulfonylurea receptor SUR1.
Diabetes. 2011 Jun;60(6):1797-804. doi: 10.2337/db10-1631. Epub 2011 May 2.
2
Direct activation of Epac by sulfonylurea is isoform selective.
Chem Biol. 2011 Feb 25;18(2):243-51. doi: 10.1016/j.chembiol.2010.12.007.
3
A specialized team approach to diagnosis and medical versus surgical treatment of infants with congenital hyperinsulinism.
Semin Pediatr Surg. 2011 Feb;20(1):32-7. doi: 10.1053/j.sempedsurg.2010.10.008.
4
Exchange protein directly activated by cyclic AMP isoform 2 is not a direct target of sulfonylurea drugs.
Assay Drug Dev Technol. 2011 Feb;9(1):88-91. doi: 10.1089/adt.2010.0338. Epub 2010 Dec 6.
5
Successful transfer to sulfonylurea therapy in an infant with developmental delay, epilepsy and neonatal diabetes (DEND) syndrome and a novel ABCC8 gene mutation.
Diabetologia. 2011 Feb;54(2):469-71. doi: 10.1007/s00125-010-1981-8. Epub 2010 Nov 26.
6
Neonatal diabetes caused by mutations in sulfonylurea receptor 1: interplay between expression and Mg-nucleotide gating defects of ATP-sensitive potassium channels.
J Clin Endocrinol Metab. 2010 Dec;95(12):E473-8. doi: 10.1210/jc.2010-1231. Epub 2010 Sep 1.
7
Paracrinology of islets and the paracrinopathy of diabetes.
Proc Natl Acad Sci U S A. 2010 Sep 14;107(37):16009-12. doi: 10.1073/pnas.1006639107. Epub 2010 Aug 26.
8
A mutation in KCNJ11 causing human hyperinsulinism (Y12X) results in a glucose-intolerant phenotype in the mouse.
Diabetologia. 2010 Nov;53(11):2352-6. doi: 10.1007/s00125-010-1866-x. Epub 2010 Aug 8.
9
Twelve type 2 diabetes susceptibility loci identified through large-scale association analysis.
Nat Genet. 2010 Jul;42(7):579-89. doi: 10.1038/ng.609.
10
Membrane potential-dependent inactivation of voltage-gated ion channels in alpha-cells inhibits glucagon secretion from human islets.
Diabetes. 2010 Sep;59(9):2198-208. doi: 10.2337/db09-1505. Epub 2010 Jun 14.
Zotero 插件