紫霉素抗性：任一核糖体亚基的改变都会影响抗生素与该亚基的结合，并且整个核糖体对该药物产生抗性。 - Suppr | 超能文献

Viomycin resistance: alterations of either ribosomal subunit affect the binding of the antibiotic to the pair subunit and the entire ribosome becomes resistant to the drug.

作者信息

Choi E C, Misumi M, Nishimura T, Tanaka N, Nomoto S, Teshima T, Shiba T

出版信息

Biochem Biophys Res Commun. 1979 Apr 13;87(3):904-10. doi: 10.1016/0006-291x(79)92043-6.

DOI:10.1016/0006-291x(79)92043-6

Abstract

摘要

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1

Viomycin resistance: alterations of either ribosomal subunit affect the binding of the antibiotic to the pair subunit and the entire ribosome becomes resistant to the drug.紫霉素抗性：任一核糖体亚基的改变都会影响抗生素与该亚基的结合，并且整个核糖体对该药物产生抗性。

Biochem Biophys Res Commun. 1979 Apr 13;87(3):904-10. doi: 10.1016/0006-291x(79)92043-6.

2

The inhibition of ribosomal translocation by viomycin.紫霉素对核糖体易位的抑制作用。

Eur J Biochem. 1977 Dec;81(3):491-7. doi: 10.1111/j.1432-1033.1977.tb11974.x.

3

Dual interference of hygromycin B with ribosomal translocation and with aminoacyl-tRNA recognition.潮霉素B对核糖体易位和氨酰tRNA识别的双重干扰。

Eur J Biochem. 1978 Jun 1;87(1):21-7. doi: 10.1111/j.1432-1033.1978.tb12347.x.

4

Similarities and differences in the inhibition patterns of thiostrepton and viomycin: evidence for two functionally different populations of P sites when occupied with AcPhe-tRNA.硫链丝菌素和紫霉素抑制模式的异同：当被乙酰苯丙氨酸 - 转运RNA占据时P位点存在两个功能不同群体的证据。

Biochim Biophys Acta. 1990 Aug 27;1050(1-3):193-6. doi: 10.1016/0167-4781(90)90165-x.

5

Induction of translational errors (misreading) by tuberactinomycins and capreomycins.结核放线菌素和卷曲霉素诱导翻译错误（错读）

Biochem Biophys Res Commun. 1980 Dec 16;97(3):1047-42. doi: 10.1016/0006-291x(80)91481-3.

6

Insights into protein biosynthesis and ribosome function through inhibitors.通过抑制剂深入了解蛋白质生物合成与核糖体功能。

Prog Nucleic Acid Res Mol Biol. 1976;17:217-45. doi: 10.1016/s0079-6603(08)60071-9.

7

Analysis of ribosomes from viomycin-sensitive and -resistant strains of Mycobacterium smegmatis.耻垢分枝杆菌对紫霉素敏感和耐药菌株核糖体的分析。

J Bacteriol. 1972 Oct;112(1):1-6. doi: 10.1128/jb.112.1.1-6.1972.

8

Molecular mechanism of viomycin inhibition of peptide elongation in bacteria.紫霉素抑制细菌中肽链延伸的分子机制。

Proc Natl Acad Sci U S A. 2016 Jan 26;113(4):978-83. doi: 10.1073/pnas.1517541113. Epub 2016 Jan 11.

9

Interaction between 30 S ribosomal components in a viomycin resistant mutant of Mycobacterium smegmatis.耻垢分枝杆菌紫霉素抗性突变体中30 S核糖体组分间的相互作用

Microbiol Immunol. 1979;23(7):595-604. doi: 10.1111/j.1348-0421.1979.tb00500.x.

10

On the structural basis of peptide-bond formation and antibiotic resistance from atomic structures of the large ribosomal subunit.基于大核糖体亚基的原子结构对肽键形成和抗生素抗性的结构基础。

FEBS Lett. 2005 Feb 7;579(4):955-8. doi: 10.1016/j.febslet.2004.11.053.

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Molecular basis for the selectivity of antituberculosis compounds capreomycin and viomycin.抗结核化合物 capreomycin 和 viomycin 选择性的分子基础。

Antimicrob Agents Chemother. 2011 Oct;55(10):4712-7. doi: 10.1128/AAC.00628-11. Epub 2011 Jul 18.

2

Mutation of tlyA confers capreomycin resistance in Mycobacterium tuberculosis.tlyA基因突变赋予结核分枝杆菌卷曲霉素耐药性。

Antimicrob Agents Chemother. 2005 Feb;49(2):571-7. doi: 10.1128/AAC.49.2.571-577.2005.

3

The translocation inhibitor tuberactinomycin binds to nucleic acids and blocks the in vitro assembly of 50S subunits.转运抑制剂结核放线菌素与核酸结合，并阻断50S亚基的体外组装。

Nucleic Acids Res. 1980 Dec 11;8(23):5767-77. doi: 10.1093/nar/8.23.5767.