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[严重少精子症作为生精小管部分梗阻的后遗症]

[Severe oligozoospermia as a sequela to partial obstruction of the seminiferous tubules].

作者信息

Schreiber G, Zollmann C, Reiber V, Zepnick H, Lauterbach H

机构信息

Klinik und Poliklinik für Hautkrankheiten, Bereiches Medizin der Friederich-Schiller-Universität Jena.

出版信息

Z Urol Nephrol. 1990 Jul;83(7):359-65.

PMID:2238877
Abstract

Arguments for the presence of a partial obstruction as cause of severe oligozoospermia may result from following findings. 1. The frequency of normal FSH values in oligozoospermia (44.9%) do not differ significantly from cases with azoospermia (50%). 2. Including a quantitative analysis of testicular morphology 5 out of 63 patients (7.9%) suffering from severe oligozoospermia show a normal spermatogenesis. 3. The frequency of this constellation in our material including severe oligozoospermia, normal testicular volume, normal FSH value, and normal spermatogenesis agree with few findings in the literature. The number of etiological factors of partial obstruction require also invasive diagnostical procedures like vas deferens radiography and surgical exploration.

摘要

认为存在部分梗阻是严重少精子症病因的观点可能源于以下发现。1. 少精子症患者中促卵泡生成素(FSH)值正常的频率(44.9%)与无精子症患者(50%)相比无显著差异。2. 对63例严重少精子症患者进行睾丸形态定量分析,其中5例(7.9%)精子发生正常。3. 在我们纳入严重少精子症、睾丸体积正常、FSH值正常及精子发生正常的研究资料中,这种情况出现的频率与文献中少数研究结果相符。部分梗阻的病因数量也需要通过输精管造影和手术探查等侵入性诊断程序来确定。

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