Consequences of NMO-IgG binding to aquaporin-4 in neuromyelitis optica.
作者信息
Rossi Andrea, Ratelade Julien, Papadopoulos Marios C, Bennett Jeffrey L, Verkman A S
出版信息
Proc Natl Acad Sci U S A. 2012 Jun 12;109(24):E1511; author reply E1512. doi: 10.1073/pnas.1203463109. Epub 2012 May 15.
Abstract
摘要
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本文引用的文献
1
Complement-dependent cytotoxicity in neuromyelitis optica requires aquaporin-4 protein assembly in orthogonal arrays.补体依赖性细胞毒性在视神经脊髓炎中需要水通道蛋白-4 蛋白在正交阵列中的组装。
J Biol Chem. 2012 Apr 20;287(17):13829-39. doi: 10.1074/jbc.M112.344325. Epub 2012 Mar 5.
2
Molecular outcomes of neuromyelitis optica (NMO)-IgG binding to aquaporin-4 in astrocytes.视神经脊髓炎免疫球蛋白 G（NMO-IgG）与星形胶质细胞水通道蛋白 4 结合的分子结果。
Proc Natl Acad Sci U S A. 2012 Jan 24;109(4):1245-50. doi: 10.1073/pnas.1109980108. Epub 2011 Nov 29.
3
Evidence against cellular internalization in vivo of NMO-IgG, aquaporin-4, and excitatory amino acid transporter 2 in neuromyelitis optica.在视神经脊髓炎中，没有证据表明 NMO-IgG、水通道蛋白-4 和兴奋性氨基酸转运体 2 会在细胞内内化。
J Biol Chem. 2011 Dec 30;286(52):45156-64. doi: 10.1074/jbc.M111.297275. Epub 2011 Nov 8.
4
Aquaporin-4 orthogonal arrays of particles are the target for neuromyelitis optica autoantibodies.水通道蛋白4颗粒正交排列是视神经脊髓炎自身抗体的靶点。
Glia. 2009 Oct;57(13):1363-73. doi: 10.1002/glia.20855.
5
Aquaporin-4-binding autoantibodies in patients with neuromyelitis optica impair glutamate transport by down-regulating EAAT2.视神经脊髓炎患者体内的水通道蛋白4结合自身抗体通过下调兴奋性氨基酸转运体2来损害谷氨酸转运。
J Exp Med. 2008 Oct 27;205(11):2473-81. doi: 10.1084/jem.20081241. Epub 2008 Oct 6.
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