成人胃出口梗阻经典生化后遗症的提示。
A reminder of the classical biochemical sequelae of adult gastric outlet obstruction.
作者信息
Ahmad Jawad, Thomson Stacey, Taylor Mark, Scoffield Julie
机构信息
Department of Surgery, Mater Hospital Belfast, Belfast, UK.
出版信息
BMJ Case Rep. 2011 Feb 2;2011:bcr0520102978. doi: 10.1136/bcr.05.2010.2978.
Abstract
The commonest cause of gastric outlet obstruction (GOO) is pyloric stenosis secondary to peptic ulcer disease or gastric carcinoma. Patients with GOO have unique metabolic sequelae, namely hypochloraemic, hypokalaemic metabolic alkalosis with paradoxical aciduria and hypocalcaemia. A case of a patient presenting as GOO is discussed. The aim of this report is to highlight the metabolic abnormalities and management in patients with GOO.
摘要
胃出口梗阻（GOO）最常见的原因是继发于消化性溃疡病或胃癌的幽门狭窄。胃出口梗阻患者有独特的代谢后遗症，即低氯性、低钾性代谢性碱中毒伴反常性酸性尿和低钙血症。本文讨论了一例表现为胃出口梗阻的患者。本报告的目的是强调胃出口梗阻患者的代谢异常及管理。
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