Roock Sophie D, Deleuze Jean-Paul, Rose Thomas, Jennes Serge, Hantson Philippe
Burn Unit, Military Hospital, Université Catholique De Louvain, Cliniques St-Luc, Brussels, Belgium.
J Emerg Trauma Shock. 2012 Apr;5(2):178-80. doi: 10.4103/0974-2700.96488.
Assault chemical burns are uncommon in northern Europe. Besides local toxicity, systemic manifestations are possible after strong acid exposure. A 40-year-old woman was admitted 1 h after a criminal assault with sulfuric acid. The total burned surface area was 35%, third degree. Injury was due to sulfuric acid (measured pH 0.9) obtained from a car battery. Immediate complications were obstructive dyspnea and metabolic acidosis. The admission arterial pH was 6.92, with total bicarbonate 8.6 mEq/l and base deficit 23.4 mEq/l. The correction of metabolic acidosis was achieved after several hours by the administration of bicarbonate and lactate buffers. The patient developed several burns-related complications (sepsis and acute renal failure). Cutaneous projections of strong acids may cause severe metabolic acidosis, particularly when copious irrigation and clothes removal cannot be immediately performed at the scene.
在北欧,攻击性化学烧伤并不常见。除了局部毒性外,接触强酸后还可能出现全身表现。一名40岁女性在遭受硫酸刑事攻击1小时后入院。烧伤总面积为35%,三度烧伤。损伤是由从汽车电池中获取的硫酸(测得pH值为0.9)所致。即刻并发症为梗阻性呼吸困难和代谢性酸中毒。入院时动脉pH值为6.92,总碳酸氢盐为8.6 mEq/l,碱缺失为23.4 mEq/l。通过给予碳酸氢盐和乳酸缓冲液,数小时后代谢性酸中毒得到纠正。患者出现了几种与烧伤相关的并发症(败血症和急性肾衰竭)。强酸的皮肤投射可能导致严重的代谢性酸中毒,尤其是在现场无法立即进行大量冲洗和脱去衣物的情况下。