Glomerular homeostasis requires a match between podocyte mass and metabolic load.
作者信息
Kopp Jeffrey B
出版信息
J Am Soc Nephrol. 2012 Aug;23(8):1273-5. doi: 10.1681/ASN.2012060618. Epub 2012 Jul 12.
Abstract
摘要
相似文献
1
Glomerular homeostasis requires a match between podocyte mass and metabolic load.肾小球内环境稳定需要足细胞数量与代谢负荷相匹配。
J Am Soc Nephrol. 2012 Aug;23(8):1273-5. doi: 10.1681/ASN.2012060618. Epub 2012 Jul 12.
2
Growth-dependent podocyte failure causes glomerulosclerosis.生长依赖性足细胞衰竭导致肾小球硬化。
J Am Soc Nephrol. 2012 Aug;23(8):1351-63. doi: 10.1681/ASN.2012030271. Epub 2012 Jul 5.
3
FSGS as an Adaptive Response to Growth-Induced Podocyte Stress.局灶节段性肾小球硬化是对生长诱导的足细胞应激的适应性反应。
J Am Soc Nephrol. 2017 Oct;28(10):2931-2945. doi: 10.1681/ASN.2017020174. Epub 2017 Jul 18.
4
ARHGEF7 (-PIX) Is Required for the Maintenance of Podocyte Architecture and Glomerular Function.ARHGEF7(-PIX)对于维持足细胞结构和肾小球功能是必需的。
J Am Soc Nephrol. 2020 May;31(5):996-1008. doi: 10.1681/ASN.2019090982. Epub 2020 Mar 18.
5
Podocyte hypertrophy, "adaptation," and "decompensation" associated with glomerular enlargement and glomerulosclerosis in the aging rat: prevention by calorie restriction.与衰老大鼠肾小球增大和肾小球硬化相关的足细胞肥大、“适应”及“失代偿”：热量限制的预防作用
J Am Soc Nephrol. 2005 Oct;16(10):2953-66. doi: 10.1681/ASN.2005050488. Epub 2005 Aug 24.
6
The Hippo pathway regulator KIBRA promotes podocyte injury by inhibiting YAP signaling and disrupting actin cytoskeletal dynamics.Hippo 通路调节剂 KIBRA 通过抑制 YAP 信号通路和破坏肌动蛋白细胞骨架动力学促进足细胞损伤。
J Biol Chem. 2017 Dec 22;292(51):21137-21148. doi: 10.1074/jbc.M117.819029. Epub 2017 Oct 5.
7
[Structure and function of podocyte].[足细胞的结构与功能]
Nihon Rinsho. 2006 Feb;64 Suppl 2:70-3.
8
[The role of podocyte damage in the pathogenesis of glomerulosclerosis and possible repair mechanisms].[足细胞损伤在肾小球硬化发病机制中的作用及可能的修复机制]
G Ital Nefrol. 2009 Nov-Dec;26(6):660-9.
9
OPN deficiency results in severe glomerulosclerosis in uninephrectomized mice.OPN 缺乏导致单侧肾切除小鼠严重的肾小球硬化。
Am J Physiol Renal Physiol. 2013 Jun 15;304(12):F1458-70. doi: 10.1152/ajprenal.00615.2012. Epub 2013 Apr 3.
10
Podocyte number and density changes during early human life.人类生命早期足细胞数量和密度的变化。
Pediatr Nephrol. 2017 May;32(5):823-834. doi: 10.1007/s00467-016-3564-5. Epub 2016 Dec 27.
引用本文的文献
1
Plasminogenuria is associated with podocyte injury, edema, and kidney dysfunction in incident glomerular disease.尿纤维蛋白溶酶原血症与初发肾小球疾病中的足细胞损伤、水肿和肾功能障碍有关。
FASEB J. 2020 Dec;34(12):16191-16204. doi: 10.1096/fj.202000413R. Epub 2020 Oct 18.
2
Podocyte injury: the role of proteinuria, urinary plasminogen, and oxidative stress.足细胞损伤：蛋白尿、尿纤溶酶原及氧化应激的作用
Am J Physiol Renal Physiol. 2016 Dec 1;311(6):F1308-F1317. doi: 10.1152/ajprenal.00162.2016. Epub 2016 Jun 22.
3
Glomerular cell cross-talk influences composition and assembly of extracellular matrix.肾小球细胞间的相互作用影响细胞外基质的组成和组装。
J Am Soc Nephrol. 2014 May;25(5):953-66. doi: 10.1681/ASN.2013070795. Epub 2014 Jan 16.
本文引用的文献
1
Growth-dependent podocyte failure causes glomerulosclerosis.生长依赖性足细胞衰竭导致肾小球硬化。
J Am Soc Nephrol. 2012 Aug;23(8):1351-63. doi: 10.1681/ASN.2012030271. Epub 2012 Jul 5.
2
Epidemiology of obesity, the metabolic syndrome, and chronic kidney disease.肥胖症、代谢综合征和慢性肾脏病的流行病学。
Curr Hypertens Rep. 2012 Apr;14(2):152-9. doi: 10.1007/s11906-012-0254-y.
3
Obesity-related glomerulopathy and podocyte injury: a mini review.肥胖相关肾小球病与足细胞损伤：一篇综述
Front Biosci (Elite Ed). 2012 Jan 1;4(3):1058-70. doi: 10.2741/E441.
4
Angiotensin II-dependent persistent podocyte loss from destabilized glomeruli causes progression of end stage kidney disease.血管紧张素 II 依赖性肾小球不稳定导致的持续足细胞丢失引起终末期肾病进展。
Kidney Int. 2012 Jan;81(1):40-55. doi: 10.1038/ki.2011.306. Epub 2011 Sep 21.
5
Metabolic syndrome and kidney disease: a systematic review and meta-analysis.代谢综合征与肾脏疾病：系统评价与荟萃分析。
Clin J Am Soc Nephrol. 2011 Oct;6(10):2364-73. doi: 10.2215/CJN.02180311. Epub 2011 Aug 18.
6
Podocyte injury damages other podocytes.足细胞损伤会损害其他足细胞。
J Am Soc Nephrol. 2011 Jul;22(7):1275-85. doi: 10.1681/ASN.2010090963. Epub 2011 Jun 30.
7
Is there an obesity-metabolic syndrome related glomerulopathy?是否存在肥胖代谢综合征相关的肾小球病？
Curr Opin Nephrol Hypertens. 2011 Jan;20(1):44-9. doi: 10.1097/MNH.0b013e3283414ca1.
8
Weight loss: a neglected intervention in the management of chronic kidney disease.体重减轻：慢性肾脏病管理中被忽视的干预措施。
Curr Opin Nephrol Hypertens. 2010 Nov;19(6):534-8. doi: 10.1097/MNH.0b013e32833f13de.
9
Scope and mechanisms of obesity-related renal disease.肥胖相关肾脏疾病的范围和机制。
Curr Opin Nephrol Hypertens. 2010 May;19(3):227-34. doi: 10.1097/MNH.0b013e3283374c09.
10
Development of focal segmental glomerulosclerosis after anabolic steroid abuse.滥用合成代谢类固醇后局灶节段性肾小球硬化症的发展。
J Am Soc Nephrol. 2010 Jan;21(1):163-72. doi: 10.1681/ASN.2009040450. Epub 2009 Nov 16.
Zotero 插件