Seaman M E
Department of Emergency Medicine, Valley Medical Center, Fresno, California 93702.
Ann Emerg Med. 1990 Jan;19(1):34-7. doi: 10.1016/s0196-0644(05)82137-7.
We present the case of a 29-year-old patient who developed a cerebral infarction, without hemorrhage, after using cocaine. A computed tomography scan of the brain at the time of initial presentation was entirely normal; a repeat CT scan on the fifth hospital day showed a basal ganglion infarction. No evidence of intracranial bleeding was detected. Theories of cocaine-induced cerebral infarction, including promotion of thrombogenesis and vasospasm, are reviewed.
我们报告一例29岁患者，其在使用可卡因后发生了无出血的脑梗死。初次就诊时的脑部计算机断层扫描完全正常；住院第5天复查CT扫描显示基底节梗死。未检测到颅内出血迹象。本文对可卡因诱发脑梗死的相关理论进行了综述，包括促进血栓形成和血管痉挛。
