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本文引用的文献

1
Response of cattle upon reexposure to Anaplasma marginale after elimination of chronic carrier infections.慢性携带者感染消除后，牛再次接触边缘无形体的反应。
Am J Vet Res. 1984 Apr;45(4):695-7.
2
Acquired cellular responsiveness in cattle cleared of Anaplasma marginale 28 months earlier.在28个月前清除边缘无浆体的牛中获得的细胞反应性。
Vet Immunol Immunopathol. 1983 Jul;4(5-6):659-63. doi: 10.1016/0165-2427(83)90072-7.
3
Common and isolate-restricted antigens of Anaplasma marginale detected with monoclonal antibodies.用单克隆抗体检测的边缘无形体的常见和菌株特异性抗原
Infect Immun. 1984 Sep;45(3):697-700. doi: 10.1128/iai.45.3.697-700.1984.
4
Cell-mediated immune responses in calves with anaplasmosis.患有无形体病的犊牛的细胞介导免疫反应。
Am J Vet Res. 1973 Jun;34(6):757-63.
5
Transmission of Anaplasma marginale Theiler by males of Dermacentor andersoni Stiles fed on an Idaho field-infected, chronic carrier cow.由取食爱达荷州野外感染的慢性带菌母牛的安德逊革蜱雄蜱传播边缘无形体（泰勒变种）。
Am J Vet Res. 1986 Oct;47(10):2269-71.
6
Detection of an Anaplasma marginale common surface protein present in all stages of infection.检测在感染各阶段均存在的边缘无形体共同表面蛋白。
J Clin Microbiol. 1986 Jun;23(6):1078-83. doi: 10.1128/jcm.23.6.1078-1083.1986.
7
Detection of Anaplasma-marginale-infected tick vectors by using a cloned DNA probe.使用克隆DNA探针检测边缘无形体感染的蜱传媒介
Proc Natl Acad Sci U S A. 1988 Feb;85(3):919-23. doi: 10.1073/pnas.85.3.919.
8
Molecular size variations in an immunoprotective protein complex among isolates of Anaplasma marginale.边缘无形体分离株中一种免疫保护蛋白复合物的分子大小变异
Infect Immun. 1988 Jun;56(6):1567-73. doi: 10.1128/iai.56.6.1567-1573.1988.
9
Detection and quantitation of Anaplasma marginale in carrier cattle by using a nucleic acid probe.利用核酸探针检测和定量带菌牛体内的边缘无浆体。
J Clin Microbiol. 1989 Feb;27(2):279-84. doi: 10.1128/jcm.27.2.279-284.1989.
10
Antigenic and genetic relatedness of eight Rickettsia tsutsugamushi antigens.八种恙虫病东方体抗原的抗原性和基因相关性
Infect Immun. 1989 Oct;57(10):3116-22. doi: 10.1128/iai.57.10.3116-3122.1989.

牛边缘无形体持续感染期间的周期性立克次体血症

Cyclic rickettsemia during persistent Anaplasma marginale infection of cattle.

作者信息

Kieser S T, Eriks I S, Palmer G H

机构信息

Department of Veterinary Microbiology and Pathology, U.S. Department of Agriculture, Pullman, Washington.

出版信息

Infect Immun. 1990 Apr;58(4):1117-9. doi: 10.1128/iai.58.4.1117-1119.1990.

DOI:10.1128/iai.58.4.1117-1119.1990
PMID:2318532
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC258591/
Abstract

Submicroscopic levels of Anaplasma marginale rickettsemia in persistently infected cattle were determined by using nucleic acid hybridization. Within individuals, the rickettsemia levels steadily increased from less than 10(4) infected erythrocytes per ml to a peak of more than 10(6) infected erythrocytes per ml and then rapidly declined. This logarithmic variation parallels the variation of the rickettsemia level seen in acute infection and suggests that cyclic emergence of antigenic variants is a mechanism of rickettsial persistence.

摘要

通过核酸杂交测定持续感染牛体内边缘无形体立克次氏体血症的亚显微水平。在个体内部，立克次氏体血症水平从每毫升少于10⁴个感染红细胞稳步上升至每毫升超过10⁶个感染红细胞的峰值，然后迅速下降。这种对数变化与急性感染中所见的立克次氏体血症水平变化相似，表明抗原变异体的周期性出现是立克次氏体持续存在的一种机制。