Scool of Psychology, Complutense University of Madrid, 28223- Madrid, Spain.
Behav Ther. 2013 Mar;44(1):62-74. doi: 10.1016/j.beth.2012.07.003. Epub 2012 Jul 23.
Previous theories and research show clear divergences on the roles of the behavioral activation system (BAS) and the behavioral inhibition system (BIS) in depression. Across four studies, we examined the effects of a sad mood on the motivational pattern of sensitivity to reward and punishment. Psychological variables associated with such changes and implications for vulnerability to depression were also explored. For this purpose, we designed a state version of the extensively used BIS/BAS Scales (Carver & White, 1994). Using samples of undergraduate students, we found that both a natural (Study 1) and a laboratory-induced sad mood (Studies 2 and 3) generated a marked decrease in sensitivity to reward but did not alter sensitivity to punishment. Study 3a showed that participants' anxious attachment predicted larger decreases in sensitivity to reward after a sad mood induction. Study 3b extended these results by showing that sensitivity to reward, when assessed after the negative mood induction, predicted increases in dysphoria 7weeks later. Implications of the results for research on vulnerability to depression are discussed.
先前的理论和研究表明,在抑郁中,行为激活系统(BAS)和行为抑制系统(BIS)的作用存在明显分歧。在四项研究中,我们考察了悲伤情绪对奖励和惩罚敏感性的动机模式的影响。还探讨了与这种变化相关的心理变量及其对抑郁易感性的影响。为此,我们设计了广泛使用的 BIS/BAS 量表(Carver & White,1994)的状态版本。使用本科生样本,我们发现,自然(研究 1)和实验室诱导的悲伤情绪(研究 2 和 3)都导致对奖励的敏感性显著降低,但对惩罚的敏感性没有改变。研究 3a 表明,参与者的焦虑依恋预测在悲伤情绪诱导后对奖励的敏感性会有更大的降低。研究 3b 通过显示在负面情绪诱导后评估的奖励敏感性,预测 7 周后抑郁情绪的增加,扩展了这些结果。讨论了这些结果对抑郁易感性研究的意义。