CrossTalk proposal: the intermittent hypoxia attending severe obstructive sleep apnoea does lead to alterations in brain structure and function.
作者信息
Gozal David
机构信息
Department of Pediatrics, Pritzker School of Medicine, Biological Sciences Division, The University of Chicago, Chicago, IL, USA.
出版信息
J Physiol. 2013 Jan 15;591(2):379-81. doi: 10.1113/jphysiol.2012.241216.
Abstract
摘要
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CrossTalk proposal: the intermittent hypoxia attending severe obstructive sleep apnoea does lead to alterations in brain structure and function.串扰假说：严重阻塞性睡眠呼吸暂停伴发的间歇性低氧确实会导致脑结构和功能的改变。
J Physiol. 2013 Jan 15;591(2):379-81. doi: 10.1113/jphysiol.2012.241216.
2
CrossTalk opposing view: the intermittent hypoxia attending severe obstructive sleep apnoea does not lead to alterations in brain structure and function.相互矛盾的观点：重度阻塞性睡眠呼吸暂停伴发的间歇性低氧不会导致脑结构和功能的改变。
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Neurocognitive function in obstructive sleep apnoea: a meta-review.阻塞性睡眠呼吸暂停的神经认知功能：荟萃分析。
Respirology. 2013 Jan;18(1):61-70. doi: 10.1111/j.1440-1843.2012.02255.x.
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High altitude exposure impairs sleep patterns, mood, and cognitive functions.高海拔暴露会影响睡眠模式、情绪和认知功能。
Psychophysiology. 2012 Sep;49(9):1298-306. doi: 10.1111/j.1469-8986.2012.01411.x. Epub 2012 Jul 16.
3
Disrupted sleep without sleep curtailment induces sleepiness and cognitive dysfunction via the tumor necrosis factor-α pathway.睡眠中断而不减少睡眠时间会通过肿瘤坏死因子-α途径引起嗜睡和认知功能障碍。
J Neuroinflammation. 2012 May 11;9:91. doi: 10.1186/1742-2094-9-91.
4
Human apolipoprotein E4 targeted replacement in mice reveals increased susceptibility to sleep disruption and intermittent hypoxia.人类载脂蛋白 E4 基因定点敲入小鼠模型揭示了对睡眠中断和间歇性低氧的易感性增加。
Am J Physiol Regul Integr Comp Physiol. 2012 Jul 1;303(1):R19-29. doi: 10.1152/ajpregu.00025.2012. Epub 2012 May 9.
5
Cognition in obstructive sleep apnea-hypopnea syndrome (OSAS): current clinical knowledge and the impact of treatment.阻塞性睡眠呼吸暂停低通气综合征（OSAS）患者的认知功能：当前的临床认识和治疗的影响。
Neuromolecular Med. 2012 Sep;14(3):180-93. doi: 10.1007/s12017-012-8182-1. Epub 2012 May 9.
6
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Cognitive function in prepubertal children with obstructive sleep apnea: a modifying role for NADPH oxidase p22 subunit gene polymorphisms?青春期前阻塞性睡眠呼吸暂停患儿的认知功能：NADPH 氧化酶 p22 亚单位基因多态性的调节作用？
Antioxid Redox Signal. 2012 Jan 15;16(2):171-7. doi: 10.1089/ars.2011.4189. Epub 2011 Oct 12.
8
Exogenous growth hormone attenuates cognitive deficits induced by intermittent hypoxia in rats.外源性生长激素可减轻间歇性低氧诱导的大鼠认知功能障碍。
Neuroscience. 2011 Nov 24;196:237-50. doi: 10.1016/j.neuroscience.2011.08.029. Epub 2011 Aug 23.
9
Sleep fragmentation induces cognitive deficits via nicotinamide adenine dinucleotide phosphate oxidase-dependent pathways in mouse.睡眠片段化通过烟酰胺腺嘌呤二核苷酸磷酸氧化酶依赖性途径诱导小鼠认知缺陷。
Am J Respir Crit Care Med. 2011 Dec 1;184(11):1305-12. doi: 10.1164/rccm.201107-1173OC. Epub 2011 Aug 25.
10
Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea.间歇性低氧诱导的认知功能障碍是通过睡眠呼吸暂停小鼠模型中的 NADPH 氧化酶活性介导的。
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