巨噬细胞在小鼠体内抗鸟分枝杆菌复合群感染的宿主防御机制中的作用。 - Suppr

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超能文献

The role of macrophages in host defence mechanisms against Mycobacterium avium complex infection induced in mice.

作者信息

Saito H, Tomioka H

机构信息

Department of Microbiology and Immunology, Shimane Medical University, Izumo, Japan.

出版信息

Res Microbiol. 1990 Feb;141(2):206-12. doi: 10.1016/0923-2508(90)90032-l.

DOI:10.1016/0923-2508(90)90032-l

PMID:2345799

Abstract

摘要

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Roles of tumour necrosis factor-alpha (TNF-alpha), transforming growth factor-beta (TGF-beta), and IL-10 in the modulation of intercellular adhesion molecule-1 (ICAM-1) expression by macrophages during mycobacterial infection.肿瘤坏死因子-α（TNF-α）、转化生长因子-β（TGF-β）和白细胞介素-10在分枝杆菌感染期间巨噬细胞对细胞间黏附分子-1（ICAM-1）表达的调节作用。

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Therapeutic effects of benzoxazinorifamycin KRM-1648 administered alone or in combination with a half-sized secretory leukocyte protease inhibitor or the nonsteroidal anti-inflammatory drug diclofenac sodium against Mycobacterium avium complex infection in mice.单独使用苯并恶嗪诺利福霉素KRM-1648或与半量分泌型白细胞蛋白酶抑制剂或非甾体抗炎药双氯芬酸钠联合使用对小鼠鸟分枝杆菌复合群感染的治疗效果。

Antimicrob Agents Chemother. 1999 Feb;43(2):360-4. doi: 10.1128/AAC.43.2.360.

H2O2 induces monocyte apoptosis and reduces viability of Mycobacterium avium-M. intracellulare within cultured human monocytes.过氧化氢可诱导单核细胞凋亡，并降低人单核细胞培养物中鸟分枝杆菌-胞内分枝杆菌的活力。

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