Strassberg D S, Mahoney J M, Schaugaard M, Hale V E
Department of Psychology, University of Utah, Salt Lake City 84112.
Arch Sex Behav. 1990 Jun;19(3):251-7. doi: 10.1007/BF01541550.
Despite the prevalence of premature ejaculation and the success in its treatment, researchers have yet to offer a compelling, empirically based theory regarding its nature or etiology. This study explored a model that argues that anxiety may not be necessary for the existence of this dysfunction. Fifteen premature ejaculators (PEs) and 17 nonpremature ejaculators (NPEs) engaged in self-stimulation to orgasm both in the laboratory and at home. The following specific hypotheses were tested: Compared to NPEs, PEs would demonstrate (i) shorter orgasmic latencies, both in the lab and at home, and (ii) equally accurate estimates of these latencies. Results offered strong support for both hypotheses. These findings, and those derived from a questionnaire completed by subjects, were seen as consistent with a psychophysiologic model of premature ejaculation. According to this model, the role of anxiety is seen as variable, interacting with the somatic vulnerability of the individual to determine orgasmic latency.
尽管早泄很常见且治疗取得了成功，但研究人员尚未提出一个令人信服的、基于实证的关于其本质或病因的理论。本研究探索了一个模型，该模型认为焦虑对于这种性功能障碍的存在可能并非必要条件。15名早泄患者（PEs）和17名非早泄患者（NPEs）在实验室和家中通过自我刺激达到性高潮。对以下具体假设进行了检验：与非早泄患者相比，早泄患者在实验室和家中都将表现出（i）更短的性高潮潜伏期，以及（ii）对这些潜伏期的估计同样准确。结果为这两个假设提供了有力支持。这些发现以及受试者填写的问卷所得出的结果，被视为与早泄的心理生理模型相一致。根据该模型，焦虑的作用被视为可变的，与个体的躯体易感性相互作用以确定性高潮潜伏期。
