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[导致前列腺尿道扩张的交界区综合征:一种新的病理实体?]

[Junctional syndrome responsible for prostatic urethral dilatation: a new pathological entity?].

作者信息

L'Hermite J, Six A, Schmitt M, Hubert J, Bretagne M C

机构信息

Service d'urologie, CHR Nancy-Brabois, Vandoeuvre-les-Nancy.

出版信息

Acta Urol Belg. 1990;58(1):123-31.

PMID:2371931
Abstract

The authors report two observations of monstrous ectasia of the prostatic urethra, in boys age 12, without any previous pathological history; discovered on at the time of major dysuria previous urinary troubles with chronic retention of 500 cl and 1.250 l respectively. In the absence of any organic obstacle, etiology relieves functional troubles. If functional explorations show a normal or hypertensive detrusor, then they discuss the possibility of vesico-sphincter dyssynergia or an obvious hypertension of the striated sphincter. From this moment onwards, purely intrinsic urethral functional trouble is plausible, all the more because the recovery was assured by treatments directed only at the urinary canal. In the first case, after surgical treatment owing to complex lesions (plastic reduction of the urethral ectasia preserving the posterior wall and respecting the striated sphincter) combined with diverticulectomies with urethral reimplantations), the patient recovered complete vesical waste removal at once, with normalisation of the flowmeter in six months. In the second case, a simpler treatment (sub-montanal urethrotomy of the principle) allowed a normalisation period of 10 months with complete vesical waste removal. The functional results are maintained after a period of nine and two years respectively. The integrity of the striated sphincter controlled post-operatively by the operatory gesture on the urinary canal and confronts the authors in the idea of intrinsic urethral pathology, although electronic microscopic examination may not have shown achalasia.

摘要

作者报告了两例12岁男孩前列腺尿道巨大扩张的病例,此前无任何病史;分别在严重排尿困难时发现,伴有慢性尿潴留,尿量分别为500毫升和1250升。在没有任何器质性梗阻的情况下,病因缓解了功能性问题。如果功能检查显示逼尿肌正常或亢进,那么他们会讨论膀胱括约肌协同失调或横纹肌括约肌明显亢进的可能性。从这时起,纯粹的尿道内在功能性问题是有可能的,尤其是因为仅针对尿道的治疗确保了恢复。在第一例中,由于复杂病变(保留后壁并尊重横纹肌括约肌的尿道扩张整形复位)并结合尿道再植的憩室切除术进行手术治疗后,患者立即恢复了完全的膀胱排空,六个月内流量计恢复正常。在第二例中,一种更简单的治疗方法(原则上的经会阴尿道切开术)使排尿正常化的时间为10个月,膀胱完全排空。分别在九年和两年后功能结果得以维持。尽管电子显微镜检查可能未显示贲门失弛缓症,但通过对尿道的手术操作术后控制横纹肌括约肌的完整性,使作者想到了尿道内在病理的问题。

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