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Effects of immunomodulators on the response induced by vaccines against autoimmune diseases.
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Remodeling of the Immune Response With Aging: Immunosenescence and Its Potential Impact on COVID-19 Immune Response.
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Immunosenescence, autoimmunity, and rheumatoid arthritis.
Exp Gerontol. 2003 Aug;38(8):833-41. doi: 10.1016/s0531-5565(03)00090-1.
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Immune receptor signaling, aging and autoimmunity.
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Immune aging - A mechanism in autoimmune disease.
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Rheumatoid arthritis: a role for immunosenescence?
J Am Geriatr Soc. 2010 Aug;58(8):1565-75. doi: 10.1111/j.1532-5415.2010.02965.x. Epub 2010 Jul 19.

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Aging-associated DNA methylation of LEF1 modulates inflammation and neurodegenerative pathways.
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Booster COVID-19 mRNA vaccination ameliorates impaired B-cell but not T-cell responses in older adults.
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Seasonality as a risk factor for deaths in Parkinson's disease.
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Inflammaging, immunosenescence, and cardiovascular aging: insights into long COVID implications.
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Inclusion body myositis, viral infections, and TDP-43: a narrative review.
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Adaptive immunity and atherosclerosis: aging at its crossroads.
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Dynamics of T-cell Responses Following COVID-19 mRNA Vaccination and Breakthrough Infection in Older Adults.
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Telomeres and age-related disease: how telomere biology informs clinical paradigms.
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IL-7- and IL-15-mediated TCR sensitization enables T cell responses to self-antigens.
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Peripheral selection rather than thymic involution explains sudden contraction in naive CD4 T-cell diversity with age.
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Decline in miR-181a expression with age impairs T cell receptor sensitivity by increasing DUSP6 activity.
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KIR2DL4 (CD158d): An activation receptor for HLA-G.
Front Immunol. 2012 Aug 20;3:258. doi: 10.3389/fimmu.2012.00258. eCollection 2012.
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Expression of NKG2D and CD107 in CD8(+) effector memory lymphocytes in Churg-Strauss syndrome.
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Signaling pathways in aged T cells - a reflection of T cell differentiation, cell senescence and host environment.
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Immune aging and autoimmunity.
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Glucocorticoids suppress selected components of the senescence-associated secretory phenotype.
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