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Targeting redox biology to reverse mitochondrial dysfunction.

作者信息

Marcinek David J, Siegel Michael P

机构信息

Departments of Radiology, Bioengineering, and Pathology. University of Washington Medical School, Seattle, WA 98195, USA;

出版信息

Aging (Albany NY). 2013 Aug;5(8):588-9. doi: 10.18632/aging.100590.

DOI:10.18632/aging.100590
PMID:23945201
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3796210/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/17b3/3796210/89efc4a65a96/aging-05-588-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/17b3/3796210/89efc4a65a96/aging-05-588-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/17b3/3796210/89efc4a65a96/aging-05-588-g001.jpg

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疲劳收缩增加小鼠骨骼肌中蛋白质 S-谷胱甘肽化占有率。
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