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本文引用的文献

1
Answering the ultimate question "what is the proximal cause of aging?".回答终极问题“衰老的近端原因是什么？”
Aging (Albany NY). 2012 Dec;4(12):861-77. doi: 10.18632/aging.100525.
2
Genetics of longevity in model organisms: debates and paradigm shifts.模式生物长寿的遗传学：争论与范式转变。
Annu Rev Physiol. 2013;75:621-44. doi: 10.1146/annurev-physiol-030212-183712. Epub 2012 Nov 26.
3
Programmatic features of aging originating in development: aging mechanisms beyond molecular damage?源于发育的衰老程序化特征：超越分子损伤的衰老机制？
FASEB J. 2012 Dec;26(12):4821-6. doi: 10.1096/fj.12-210872. Epub 2012 Sep 10.
4
How to save Medicare: the anti-aging remedy.如何拯救医疗保险：抗衰老疗法。
Aging (Albany NY). 2012 Aug;4(8):547-52. doi: 10.18632/aging.100479.
5
Alternative Perspectives on Aging in Caenorhabditis elegans: Reactive Oxygen Species or Hyperfunction?秀丽隐杆线虫衰老研究的新视角：活性氧还是功能亢进？
Antioxid Redox Signal. 2013 Jul 20;19(3):321-9. doi: 10.1089/ars.2012.4840. Epub 2012 Sep 24.
6
When a theory of aging ages badly.当衰老理论过时。
Cell Mol Life Sci. 2010 Jan;67(1):1-8. doi: 10.1007/s00018-009-0138-8.
7
Against the oxidative damage theory of aging: superoxide dismutases protect against oxidative stress but have little or no effect on life span in Caenorhabditis elegans.针对衰老的氧化损伤理论：超氧化物歧化酶可抵御氧化应激，但对秀丽隐杆线虫的寿命几乎没有影响。
Genes Dev. 2008 Dec 1;22(23):3236-41. doi: 10.1101/gad.504808.
8
Remarkable longevity and stress resistance of nematode PI3K-null mutants.线虫PI3K基因缺失突变体的显著长寿和抗逆性。
Aging Cell. 2008 Jan;7(1):13-22. doi: 10.1111/j.1474-9726.2007.00348.x. Epub 2007 Nov 7.
9
Aging and immortality: quasi-programmed senescence and its pharmacologic inhibition.衰老与不朽：准程序性衰老及其药物抑制
Cell Cycle. 2006 Sep;5(18):2087-102. doi: 10.4161/cc.5.18.3288. Epub 2006 Sep 15.

Damage-induced aging and perpetual motion.

作者信息

Blagosklonny Mikhail V

机构信息

Department of Cell Stress Biology; Roswell Park Cancer Institute; Buffalo, NY USA.

出版信息

Cell Cycle. 2013 Sep 1;12(17):2709-10. doi: 10.4161/cc.26015. Epub 2013 Aug 5.

DOI:10.4161/cc.26015
PMID:23966155
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3899178/
Abstract
摘要