FACT and H3.3: new markers for the somatic hypermutation.
作者信息
Aida Masatoshi, Honjo Tasuku
机构信息
Department of Immunology and Genetic Medicine; Graduate School of Medicine; Kyoto University; Kyoto, Japan.
出版信息
Cell Cycle. 2013 Sep 15;12(18):2923-4. doi: 10.4161/cc.26178. Epub 2013 Aug 23.
Abstract
摘要
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Accumulation of the FACT complex, as well as histone H3.3, serves as a target marker for somatic hypermutation.FACT 复合体的积累以及组蛋白 H3.3 是体细胞超突变的靶标标记物。
Proc Natl Acad Sci U S A. 2013 May 7;110(19):7784-9. doi: 10.1073/pnas.1305859110. Epub 2013 Apr 22.
2
Transcription-coupled eviction of histones H2A/H2B governs V(D)J recombination.转录耦联的组蛋白 H2A/H2B 驱逐调控 V(D)J 重组。
EMBO J. 2013 May 15;32(10):1381-92. doi: 10.1038/emboj.2013.42. Epub 2013 Mar 5.
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Evolutionary comparison of the mechanism of DNA cleavage with respect to immune diversity and genomic instability.进化比较 DNA 切割机制与免疫多样性和基因组不稳定性的关系。
Biochemistry. 2012 Jul 3;51(26):5243-56. doi: 10.1021/bi3005895. Epub 2012 Jun 19.
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Nonimmunoglobulin target loci of activation-induced cytidine deaminase (AID) share unique features with immunoglobulin genes.激活诱导的胞嘧啶脱氨酶（AID）的非免疫球蛋白靶标与免疫球蛋白基因具有独特的特征。
Proc Natl Acad Sci U S A. 2012 Feb 14;109(7):2479-84. doi: 10.1073/pnas.1120791109. Epub 2012 Jan 30.
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Histone3 lysine4 trimethylation regulated by the facilitates chromatin transcription complex is critical for DNA cleavage in class switch recombination.组蛋白 3 赖氨酸 4 三甲基化受促进染色质转录复合物的调节，对于类别转换重组中的 DNA 切割至关重要。
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Activation-induced cytidine deaminase targets DNA at sites of RNA polymerase II stalling by interaction with Spt5.激活诱导的胞苷脱氨酶通过与Spt5相互作用，靶向RNA聚合酶II停滞位点的DNA。
Cell. 2010 Oct 1;143(1):122-33. doi: 10.1016/j.cell.2010.09.017.
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AID-induced decrease in topoisomerase 1 induces DNA structural alteration and DNA cleavage for class switch recombination.AID 诱导的拓扑异构酶 1 减少导致 DNA 结构改变和 DNA 断裂，从而促进类别转换重组。
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Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme.类别转换重组和高频突变需要激活诱导的胞嘧啶脱氨酶（AID），一种潜在的RNA编辑酶。
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