相似文献
1
When host defense goes awry: Modeling sepsis-induced immunosuppression.
Drug Discov Today Dis Models. 2012;9(1):e33-e38. doi: 10.1016/j.ddmod.2011.09.001.
2
Molecular and cellular aspects of sepsis-induced immunosuppression.
J Mol Med (Berl). 2008 May;86(5):495-506. doi: 10.1007/s00109-007-0300-4. Epub 2008 Feb 8.
3
The Role of IFN-β during the Course of Sepsis Progression and Its Therapeutic Potential.
Front Immunol. 2017 May 8;8:493. doi: 10.3389/fimmu.2017.00493. eCollection 2017.
4
Pathological alteration and therapeutic implications of sepsis-induced immune cell apoptosis.
Cell Death Dis. 2019 Oct 14;10(10):782. doi: 10.1038/s41419-019-2015-1.
5
Loss of suppression of tumorigenicity 2 (ST2) gene reverses sepsis-induced inhibition of lung host defense in mice.
Am J Respir Crit Care Med. 2011 Apr 1;183(7):932-40. doi: 10.1164/rccm.201006-0934OC. Epub 2010 Oct 19.
6
Design of Cecal Ligation and Puncture and Intranasal Infection Dual Model of Sepsis-Induced Immunosuppression.
J Vis Exp. 2019 Jun 15(148). doi: 10.3791/59386.
7
New perspectives on immunomodulatory therapy for bacteraemia and sepsis.
Int J Antimicrob Agents. 2010 Dec;36 Suppl 2:S70-3. doi: 10.1016/j.ijantimicag.2010.11.008. Epub 2010 Dec 3.
8
Distinct pathophysiologic mechanisms of septic acute kidney injury: role of immune suppression and renal tubular cell apoptosis in murine model of septic acute kidney injury.
Crit Care Med. 2012 Nov;40(11):2997-3006. doi: 10.1097/CCM.0b013e31825b912d.
9
MicroRNA-155 attenuates late sepsis-induced cardiac dysfunction through JNK and β-arrestin 2.
Oncotarget. 2017 Jul 18;8(29):47317-47329. doi: 10.18632/oncotarget.17636.
10
Ketoprofen impairs immunosuppression induced by severe sepsis and reveals an important role for prostaglandin E2.
Shock. 2012 Dec;38(6):620-9. doi: 10.1097/SHK.0b013e318272ff8a.
引用本文的文献
1
Vitamin D receptor (VDR) on the cell membrane of mouse macrophages participates in the formation of lipopolysaccharide tolerance: mVDR is related to the effect of artesunate to reverse LPS tolerance.
Cell Commun Signal. 2023 May 29;21(1):124. doi: 10.1186/s12964-023-01137-w.
2
Of mice and men: Laboratory murine models for recapitulating the immunosuppression of human sepsis.
Front Immunol. 2022 Aug 5;13:956448. doi: 10.3389/fimmu.2022.956448. eCollection 2022.
3
Emodin Ameliorates Intestinal Dysfunction by Maintaining Intestinal Barrier Integrity and Modulating the Microbiota in Septic Mice.
Mediators Inflamm. 2022 May 29;2022:5026103. doi: 10.1155/2022/5026103. eCollection 2022.
4
MAR1 suppresses inflammatory response in LPS-induced RAW 264.7 macrophages and human primary peripheral blood mononuclear cells via the SIRT1/PGC-1α/PPAR-γ pathway.
J Inflamm (Lond). 2021 Feb 8;18(1):8. doi: 10.1186/s12950-021-00271-x.
5
Inhibition of PTP1B Promotes M2 Polarization via MicroRNA-26a/MKP1 Signaling Pathway in Murine Macrophages.
Front Immunol. 2019 Aug 14;10:1930. doi: 10.3389/fimmu.2019.01930. eCollection 2019.
6
Modulation of the sigma-1 receptor-IRE1 pathway is beneficial in preclinical models of inflammation and sepsis.
Sci Transl Med. 2019 Feb 6;11(478). doi: 10.1126/scitranslmed.aau5266.
7
Qiang-Xin 1 Formula Prevents Sepsis-Induced Apoptosis in Murine Cardiomyocytes by Suppressing Endoplasmic Reticulum- and Mitochondria-Associated Pathways.
Front Pharmacol. 2018 Jul 30;9:818. doi: 10.3389/fphar.2018.00818. eCollection 2018.
8
Surgical and immune reconstitution murine models in bone marrow research: Potential for exploring mechanisms in sepsis, trauma and allergy.
World J Exp Med. 2017 Aug 20;7(3):58-77. doi: 10.5493/wjem.v7.i3.58.
本文引用的文献
1
Deaths: preliminary data for 2009.
Natl Vital Stat Rep. 2011 Mar;59(4):1-51.
2
Endotoxin tolerance represents a distinctive state of alternative polarization (M2) in human mononuclear cells.
J Immunol. 2011 Jun 15;186(12):7243-54. doi: 10.4049/jimmunol.1001952. Epub 2011 May 16.
3
Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients.
Crit Care. 2011;15(2):R99. doi: 10.1186/cc10112. Epub 2011 Mar 21.
4
Dose-dependent effect of anti-CTLA-4 on survival in sepsis.
Shock. 2011 Jul;36(1):38-44. doi: 10.1097/SHK.0b013e3182168cce.
5
Upregulation of programmed death-1 on T cells and programmed death ligand-1 on monocytes in septic shock patients.
Crit Care. 2011;15(1):R70. doi: 10.1186/cc10059. Epub 2011 Feb 24.
6
Colon ascendens stent peritonitis (CASP)--a standardized model for polymicrobial abdominal sepsis.
J Vis Exp. 2010 Dec 18(46):2299. doi: 10.3791/2299.
7
Modulation of dendritic cell differentiation in the bone marrow mediates sustained immunosuppression after polymicrobial sepsis.
J Immunol. 2011 Jan 15;186(2):977-86. doi: 10.4049/jimmunol.1001147. Epub 2010 Dec 15.
8
PD-L1 blockade improves survival in experimental sepsis by inhibiting lymphocyte apoptosis and reversing monocyte dysfunction.
Crit Care. 2010;14(6):R220. doi: 10.1186/cc9354. Epub 2010 Nov 30.
9
Loss of suppression of tumorigenicity 2 (ST2) gene reverses sepsis-induced inhibition of lung host defense in mice.
Am J Respir Crit Care Med. 2011 Apr 1;183(7):932-40. doi: 10.1164/rccm.201006-0934OC. Epub 2010 Oct 19.
10
Differential regulation of the PGC family of genes in a mouse model of Staphylococcus aureus sepsis.
PLoS One. 2010 Jul 15;5(7):e11606. doi: 10.1371/journal.pone.0011606.
Zotero 插件