Suppressor of cytokine signaling 3 expression is diminished in sinonasal tissues from patients with chronic rhinosinusitis with nasal polyps.
作者信息
Hulse Kathryn E, Chaung Katrina, Seshadri Sudarshan, Suh Lydia, Norton James E, Carter Roderick G, Kern Robert C, Conley David B, Chandra Rakesh K, Tan Bruce K, Peters Anju T, Grammer Leslie C, Harris Kathleen E, Torgerson Troy R, Kato Atsushi, Schleimer Robert P
机构信息
Division of Allergy-Immunology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Ill.
Department of Otolaryngology, Northwestern University Feinberg School of Medicine, Chicago, Ill.
出版信息
J Allergy Clin Immunol. 2014 Jan;133(1):275-277.e1. doi: 10.1016/j.jaci.2013.08.015. Epub 2013 Sep 29.
Abstract
摘要
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本文引用的文献
1
Regional differences in the expression of innate host defense molecules in sinonasal mucosa.鼻窦黏膜中固有宿主防御分子表达的区域差异。
J Allergy Clin Immunol. 2013 Nov;132(5):1227-1230.e5. doi: 10.1016/j.jaci.2013.05.042. Epub 2013 Jul 26.
2
Chronic rhinosinusitis with nasal polyps is characterized by B-cell inflammation and EBV-induced protein 2 expression.伴有鼻息肉的慢性鼻-鼻窦炎的特征是 B 细胞炎症和 EBV 诱导蛋白 2 的表达。
J Allergy Clin Immunol. 2013 Apr;131(4):1075-83, 1083.e1-7. doi: 10.1016/j.jaci.2013.01.043. Epub 2013 Mar 6.
3
Chronic rhinosinusitis with polyps and without polyps is associated with increased expression of suppressors of cytokine signaling 1 and 3.伴有息肉和不伴息肉的慢性鼻-鼻窦炎与细胞因子信号转导抑制因子 1 和 3 的表达增加有关。
J Allergy Clin Immunol. 2013 Mar;131(3):772-80. doi: 10.1016/j.jaci.2012.12.671. Epub 2013 Jan 30.
4
Suppression of cytokine signaling by SOCS3: characterization of the mode of inhibition and the basis of its specificity.SOCS3 通过抑制细胞因子信号通路：抑制模式的特征及其特异性的基础。
Immunity. 2012 Feb 24;36(2):239-50. doi: 10.1016/j.immuni.2011.12.015.
5
Evidence for altered activity of the IL-6 pathway in chronic rhinosinusitis with nasal polyps.慢性鼻息肉鼻窦炎患者白细胞介素 6 通路活性改变的证据。
J Allergy Clin Immunol. 2010 Feb;125(2):397-403.e10. doi: 10.1016/j.jaci.2009.10.072.
6
Perspectives on the etiology of chronic rhinosinusitis.慢性鼻窦炎病因学的观点。
Curr Opin Otolaryngol Head Neck Surg. 2010 Feb;18(1):21-6. doi: 10.1097/MOO.0b013e3283350053.
7
Different types of T-effector cells orchestrate mucosal inflammation in chronic sinus disease.不同类型的T效应细胞在慢性鼻窦炎中协调黏膜炎症。
J Allergy Clin Immunol. 2008 Nov;122(5):961-8. doi: 10.1016/j.jaci.2008.07.008. Epub 2008 Sep 19.
8
Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced T(H)17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome.高IgE综合征中新型信号转导及转录激活因子3（STAT3）突变、辅助性T细胞17（TH17）细胞数量减少以及STAT3磷酸化存在不同程度缺陷
J Allergy Clin Immunol. 2008 Jul;122(1):181-7. doi: 10.1016/j.jaci.2008.04.037.
9
Evidence of a role for B cell-activating factor of the TNF family in the pathogenesis of chronic rhinosinusitis with nasal polyps.肿瘤坏死因子家族B细胞激活因子在伴鼻息肉慢性鼻-鼻窦炎发病机制中的作用证据。
J Allergy Clin Immunol. 2008 Jun;121(6):1385-92, 1392.e1-2. doi: 10.1016/j.jaci.2008.03.002. Epub 2008 Apr 14.
10
Rhinosinusitis: establishing definitions for clinical research and patient care.鼻窦炎：为临床研究和患者护理确立定义。
J Allergy Clin Immunol. 2004 Dec;114(6 Suppl):155-212. doi: 10.1016/j.jaci.2004.09.029.
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