Hellström S, Goldie P, Salén B, Stenfors L E
Am J Otolaryngol. 1985 May-Jun;6(3):220-2. doi: 10.1016/s0196-0709(85)80089-2.
In an animal model, a stream of chilled air blown into the external auditory canal evoked an edematous pars flaccida and accumulation of a serous effusion in the attic. The degree of inflammatory changes depended on the temperature of the stimuli. The nervous system appears to mediate the observed changes, as the vagus nerve, representing the parasympathetic system, potentiates vascular leakage, and the sympathetic nerves inhibit leakage.
在动物模型中，一股吹入外耳道的冷空气会引起松弛部水肿和上鼓室浆液性积液的积聚。炎症变化的程度取决于刺激的温度。神经系统似乎介导了所观察到的变化，因为代表副交感神经系统的迷走神经会增强血管渗漏，而交感神经则抑制渗漏。
