Goldie P, Hellström S
Department of Anatomy, University of Umeå, Sweden.
Acta Otolaryngol. 1988 Jul-Aug;106(1-2):10-8. doi: 10.3109/00016488809107365.
The production of middle ear effusion by mechanical stimulation was studied in sympathectomized and vagotomized animals. Mechanical stimulation was obtained by a 14 degree C airstream in the external auditory canal. In sympathectomized animals this procedure caused effusion production and vasoconstriction of pars flaccida vessels. Mechanical stimulation in the vagotomized animals did not cause any production of effusion material. Degranulated mast cells were observed in all animals, including controls. The study indicates that the vagal nerve, in contrast to the sympathetic nerves, is involved in the mechanisms causing the leakage of fluid into the middle ear cavity, when cold air is blown into the external auditory canal.
在交感神经切除和迷走神经切断的动物中研究了机械刺激引起的中耳积液。通过在外耳道中通入14摄氏度的气流来实现机械刺激。在交感神经切除的动物中，该操作导致了积液产生以及松弛部血管的血管收缩。在迷走神经切断的动物中，机械刺激未引起任何积液物质的产生。在所有动物（包括对照组）中均观察到了脱颗粒的肥大细胞。该研究表明，与交感神经不同，当冷空气吹入外耳道时，迷走神经参与了导致液体漏入中耳腔的机制。
