吉兰-巴雷综合征并发罕见的电解质紊乱。
Uncommon dyselectrolytemia complicating Guillain-Barré syndrome.
作者信息
Saroja Aralikatte Onkarappa, Naik Karkal Ravishankar, Khanpet Mallikarjun S
机构信息
Department of Neurology, KLE University's Jawaharlal Nehru Medical College and KLES', Dr. Prabhakar Kore Hospital and MRC, Nehrunagar, Belgaum - 590 010, India.
出版信息
J Neurosci Rural Pract. 2013 Jul;4(3):328-30. doi: 10.4103/0976-3147.118794.
Guillain-Barré syndrome (GBS) and hypokalemic paralysis are common causes of acute flaccid quadriparesis and specific therapeutic interventions differ. Simultaneous occurrence of severe hypokalemia in patients with GBS at the time of presentation can cause diagnostic and therapeutic dilemma. Presence of hypomagnesemia with hypokalemia in patients with GBS can be perplexing and pose further challenges. Evaluation for preexisting inherited or other associated metabolic disturbances is needed in the presence of such complex dyselectrolytemia. We report the rare association of GBS with severe hypokalemia and hypomagnesemia in a 41-year-old male presenting with acute flaccid quadriparesis and the therapeutic challenges faced.
吉兰-巴雷综合征（GBS）和低钾性麻痹是急性弛缓性四肢瘫的常见病因，且具体治疗干预措施有所不同。GBS患者在发病时同时出现严重低钾血症可导致诊断和治疗困境。GBS患者合并低镁血症和低钾血症可能令人困惑，并带来更多挑战。在存在这种复杂的电解质紊乱时，需要评估是否存在既往遗传性或其他相关代谢紊乱。我们报告了一名41岁男性，以急性弛缓性四肢瘫就诊，其罕见地合并了GBS与严重低钾血症和低镁血症，以及所面临的治疗挑战。
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