Kuroda Masasko, Nishida Akihiro, Kikuchi Masashi, Kurimoto Yasuo
Department of Ophthalmology, Kobe City Medical Center General Hospital, Kobe, Hyogo, Japan.
Clin Ophthalmol. 2013;7:2235-7. doi: 10.2147/OPTH.S54231. Epub 2013 Nov 14.
We report the case of a 66-year-old Japanese man who developed neovascular glaucoma secondary to Purtscher's retinopathy following a head injury. The patient presented at our hospital with blurred vision and a visual field abnormality in his left eye 1 month after suffering from a head injury. Upon initial presentation, his best-corrected visual acuity on a decimal chart was 1.5 oculus dexter and 0.6 oculus sinister. The intraocular pressure (IOP) was 12 mmHg in both eyes. Fundus examination of the left eye revealed multiple white lesions in the posterior pole. Optical coherence tomography demonstrated retinal edema, particularly in the inner retina. On the basis of these findings, a diagnosis of Purtscher's retinopathy was made. One month after the initial examination, the visual acuity in the left eye deteriorated to 0.01 in decimal chart, and the IOP increased to 37 mmHg. Gonioscopy showed angle neovascularization. The patient received an intravitreal bevacizumab injection and panretinal photocoagulation. Subsequently, the IOP normalized and the angle neovascularization regressed.
我们报告了一例66岁的日本男性病例,该患者在头部受伤后发生了继发于普尔夏视网膜病变的新生血管性青光眼。患者在头部受伤1个月后因左眼视力模糊和视野异常前来我院就诊。初诊时,他在十进制视力表上的最佳矫正视力右眼为1.5,左眼为0.6。双眼眼压(IOP)均为12 mmHg。左眼眼底检查发现后极部有多个白色病变。光学相干断层扫描显示视网膜水肿,尤其是内层视网膜。基于这些发现,诊断为普尔夏视网膜病变。初次检查1个月后,左眼视力在十进制视力表上恶化至0.01,眼压升至37 mmHg。前房角镜检查显示房角新生血管形成。患者接受了玻璃体内贝伐单抗注射和全视网膜光凝治疗。随后,眼压恢复正常,房角新生血管消退。