Toll样受体（TLR）-3：类风湿关节炎背后的强大驱动力。 - Suppr

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超能文献

Toll-like receptor (TLR)-3: a potent driving force behind rheumatoid arthritis.

作者信息

Li Xiaofeng, Xu Tao, Wang Yarui, Huang Cheng, Li Jun

机构信息

School of Pharmacy, Anhui Medical University, Mei Shan Road, Hefei, 230032, Anhui Province, China.

出版信息

Clin Rheumatol. 2014 Feb;33(2):291-2. doi: 10.1007/s10067-013-2418-9. Epub 2013 Nov 21.

DOI:10.1007/s10067-013-2418-9

PMID:24258070

Abstract

摘要

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Toll-like receptor (TLR)-3: a potent driving force behind rheumatoid arthritis.Toll样受体（TLR）-3：类风湿关节炎背后的强大驱动力。

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Inflammatory immune cell responses and Toll-like receptor expression in synovial tissues in rheumatoid arthritis patients treated with biologics or DMARDs.生物制剂或 DMARDs 治疗的类风湿关节炎患者滑膜组织中的炎症免疫细胞反应和 Toll 样受体表达。

Clin Rheumatol. 2013 Jun;32(6):853-61. doi: 10.1007/s10067-013-2209-3. Epub 2013 Feb 10.

Toll-like receptor--a potent driving force behind rheumatoid arthritis.Toll样受体——类风湿关节炎背后的强大驱动力。

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Can serum interleukin-17 and interleukin-10 levels predict response to biologic treatments in patients with rheumatoid arthritis?血清白细胞介素-17和白细胞介素-10水平能否预测类风湿关节炎患者对生物治疗的反应？

Rheumatology (Oxford). 2019 Oct 1;58(10):1872-1873. doi: 10.1093/rheumatology/kez147.

Targeting interleukin-33 in rheumatoid arthritis.靶向类风湿关节炎中的白细胞介素-33

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Chemokine secretion of rheumatoid arthritis synovial fibroblasts stimulated by Toll-like receptor 2 ligands.Toll样受体2配体刺激类风湿性关节炎滑膜成纤维细胞的趋化因子分泌

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The expression of toll-like receptors 3 and 7 in rheumatoid arthritis synovium is increased and costimulation of toll-like receptors 3, 4, and 7/8 results in synergistic cytokine production by dendritic cells.类风湿性关节炎滑膜中Toll样受体3和7的表达增加，Toll样受体3、4和7/8的共刺激导致树突状细胞协同产生细胞因子。

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Tumor necrosis factor alpha blockade treatment down-modulates the increased systemic and local expression of Toll-like receptor 2 and Toll-like receptor 4 in spondylarthropathy.肿瘤坏死因子α阻断治疗可下调脊柱关节病中Toll样受体2和Toll样受体4全身及局部表达的增加。

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Interleukin-17 increases the expression of Toll-like receptor 3 via the STAT3 pathway in rheumatoid arthritis fibroblast-like synoviocytes.白细胞介素-17 通过 STAT3 通路增加类风湿关节炎成纤维样滑膜细胞中 Toll 样受体 3 的表达。

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[Adhesion molecule in pathogenesis of rheumatoid arthritis].[黏附分子在类风湿关节炎发病机制中的作用]

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Front Immunol. 2022 Mar 3;13:812774. doi: 10.3389/fimmu.2022.812774. eCollection 2022.

Innate immunity drives pathogenesis of rheumatoid arthritis.先天免疫导致类风湿关节炎的发病机制。

Biomed J. 2021 Apr;44(2):172-182. doi: 10.1016/j.bj.2020.06.010. Epub 2020 Jul 8.

本文引用的文献

Clin Rheumatol. 2013 Jun;32(6):853-61. doi: 10.1007/s10067-013-2209-3. Epub 2013 Feb 10.

Arthritis is associated with T-cell-induced upregulation of Toll-like receptor 3 on synovial fibroblasts.关节炎与 T 细胞诱导的滑膜成纤维细胞 Toll 样受体 3 的上调有关。

Arthritis Res Ther. 2011 Jun 27;13(3):R103. doi: 10.1186/ar3384.

Engagement of toll-like receptor 3 induces vascular endothelial growth factor and interleukin-8 in human rheumatoid synovial fibroblasts.Toll 样受体 3 的激活诱导人类风湿滑膜成纤维细胞血管内皮生长因子和白细胞介素 8 的产生。

Korean J Intern Med. 2010 Dec;25(4):429-35. doi: 10.3904/kjim.2010.25.4.429. Epub 2010 Nov 27.

Overexpression of toll-like receptors 3 and 4 in synovial tissue from patients with early rheumatoid arthritis: toll-like receptor expression in early and longstanding arthritis.早期类风湿性关节炎患者滑膜组织中Toll样受体3和4的过表达：早期和长期关节炎中的Toll样受体表达

Arthritis Rheum. 2008 Dec;58(12):3684-92. doi: 10.1002/art.24140.

Type I interferons might form the link between Toll-like receptor (TLR) 3/7 and TLR4-mediated synovial inflammation in rheumatoid arthritis (RA).I型干扰素可能是类风湿性关节炎（RA）中Toll样受体（TLR）3/7与TLR4介导的滑膜炎症之间的联系。

Ann Rheum Dis. 2009 Sep;68(9):1486-93. doi: 10.1136/ard.2007.086421. Epub 2008 Sep 2.

Arthritis Rheum. 2005 Aug;52(8):2313-22. doi: 10.1002/art.21278.

Expression of toll-like receptors 2 and 4 in rheumatoid synovial tissue and regulation by proinflammatory cytokines interleukin-12 and interleukin-18 via interferon-gamma.类风湿性滑膜组织中Toll样受体2和4的表达以及促炎细胞因子白细胞介素-12和白细胞介素-18通过干扰素-γ的调节作用

Arthritis Rheum. 2004 Dec;50(12):3856-65. doi: 10.1002/art.20678.

Expression of Toll-like receptor 2 on CD16+ blood monocytes and synovial tissue macrophages in rheumatoid arthritis.类风湿关节炎中CD16⁺血液单核细胞和滑膜组织巨噬细胞上Toll样受体2的表达

Arthritis Rheum. 2004 May;50(5):1457-67. doi: 10.1002/art.20219.

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