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一种用于高度预防起搏器介导性心动过速的新算法。

A new algorithm for a high level of protection against pacemaker-mediated tachycardia.

作者信息

Lamaison D, Girodo S, Limousin M

机构信息

Chu Saint-Jacques, Clermont-Ferrand, France.

出版信息

Pacing Clin Electrophysiol. 1988 Nov;11(11 Pt 2):1715-21. doi: 10.1111/j.1540-8159.1988.tb06301.x.

Abstract

Pacemaker-mediated tachycardia (PMT) is a well-known complication of DDD pacing. PMT needs a permeable V-A conduction and is usually initiated by a premature ventricular or atrial systole, artifact sensing, or misprogramming (long AV delay [AVD]). Today, protection against PMT is expected from pacemaker multiprogrammability. Unfortunately, this prevention is often ineffective; postventricular atrial refractory period (PVARP) must be prolonged, which limits the upper tracking rate and the patient capacity. The new Chorus ELA Medical DDD pacemaker provides classic protection against PMT (PVARP prolongation after premature V or A complex, magnet application, noise sensing), but also automatically reduces an eventual PMT and adjusts AVD or PVARP for a high level of protection. The process is divided in four steps: (1) a sensing step for 16 cycles, with V-P conduction analysis; (2) confirmation of the presence of the PMT and analysis of V-A conduction time; (3) a termination step, by extending the PVARP after the following ventricular heart beat; and (4) in case of immediate recurrence of the tachycardia, reprogramming of the AVD and eventually of the PVARP. By first reducing AVD, before reprogramming PVARP, the pacemaker preserves point 2:1, providing a higher exercise capacity. This algorithm was successfully tested in three patients who had a permeable V-A conduction, without any adverse effect.

摘要

起搏器介导的心动过速(PMT)是双腔(DDD)起搏一种众所周知的并发症。PMT需要存在可传导的室房(V-A)传导,通常由室性或房性早搏、伪信号感知或程控错误(长房室延迟[AVD])引发。如今,期望通过起搏器的多功能程控来预防PMT。不幸的是,这种预防措施常常无效;必须延长心室后心房不应期(PVARP),这限制了上限跟踪频率和患者的耐受性。新型伊索普(Chorus)ELA Medical DDD起搏器不仅提供了针对PMT的经典保护措施(室性或房性早搏后、施加磁铁、噪声感知后延长PVARP),还能自动终止可能发生的PMT,并调整AVD或PVARP以实现高水平的保护。该过程分为四个步骤:(1)进行16个心动周期的感知步骤,并分析室房(V-A)传导;(2)确认是否存在PMT并分析室房(V-A)传导时间;(3)通过在下一个心室搏动后延长PVARP来终止心动过速;(4)如果心动过速立即复发,则重新程控AVD,并最终重新程控PVARP。通过在重新程控PVARP之前先缩短AVD,起搏器维持了2:1的比例,从而提供了更高的运动能力。该算法已在三名存在可传导室房(V-A)传导的患者中成功测试,且未产生任何不良影响。

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