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良性前列腺增生患者的梗阻性尿路病

Obstructive uropathy in patients with benign prostatic hyperplasia.

作者信息

Sarmina I, Resnick M I

机构信息

Division of Urology, Case Western Reserve University School of Medicine, Cleveland, Ohio.

出版信息

J Urol. 1989 Apr;141(4):866-9. doi: 10.1016/s0022-5347(17)41035-4.

DOI:10.1016/s0022-5347(17)41035-4
PMID:2467012
Abstract

In an attempt to determine the role of obstructive prostatic hyperplasia as a cause of irreversible renal function the records of 32 men with a diagnosis of benign prostatic hyperplasia and renal failure were reviewed. Patients were classified as either having a serum creatinine of more than or less than 4 mg.per 100 ml. when they were discharged from the hospital. Admission serum creatinine levels were comparable in both groups. Characteristics of the group with permanently impaired renal function included a longer mean duration of symptoms, higher incidence of urinary tract infection, lower average volume of residual urine and greater incidence of small kidneys with increased parenchymal echogenicity. These data assist in the objective prediction of patients who will have irreversible renal dysfunction when presenting with obstructive benign prostatic hyperplasia.

摘要

为了确定梗阻性前列腺增生作为不可逆肾功能病因的作用,对32例诊断为良性前列腺增生和肾衰竭的男性患者的记录进行了回顾。患者出院时血清肌酐大于或小于4mg/100ml进行分类。两组患者入院时血清肌酐水平相当。肾功能永久受损组的特征包括平均症状持续时间更长、尿路感染发生率更高、残余尿量平均体积更低以及实质回声增强的小肾脏发生率更高。这些数据有助于客观预测患有梗阻性良性前列腺增生时将出现不可逆肾功能障碍的患者。

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1
Obstructive uropathy in patients with benign prostatic hyperplasia.良性前列腺增生患者的梗阻性尿路病
J Urol. 1989 Apr;141(4):866-9. doi: 10.1016/s0022-5347(17)41035-4.
2
Prostatic enlargement.前列腺肿大
Practitioner. 1989 Apr 8;233(1466):512-7.
3
Risk factors for lower urinary tract symptoms suggestive of benign prostatic hyperplasia in a community based population of healthy aging men: the Krimpen Study.基于社区的健康老年男性人群中提示良性前列腺增生的下尿路症状的危险因素:克林彭研究
J Urol. 2009 Feb;181(2):710-6. doi: 10.1016/j.juro.2008.10.025. Epub 2008 Dec 16.
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Influence of bladder outlet obstruction and detrusor contractility on residual urine in patients with benign prostatic hyperplasia.膀胱出口梗阻和逼尿肌收缩力对良性前列腺增生患者残余尿量的影响。
Chin Med J (Engl). 2003 Oct;116(10):1508-10.
5
Urodynamic risk factors for renal dysfunction in men with obstructive and nonobstructive voiding dysfunction.梗阻性和非梗阻性排尿功能障碍男性肾功能不全的尿动力学危险因素。
J Urol. 1997 Jul;158(1):181-5. doi: 10.1097/00005392-199707000-00059.
6
Urinary flow rate in benign prostatic hypertrophy.
Int Surg. 1976 Feb;61(2):109-11.
7
Benign prostatic hyperplasia (BHP) and bothersome voiding symptoms.良性前列腺增生(BHP)与令人烦恼的排尿症状。
J Urol (Paris). 1995;101(1):7-9.
8
Serum creatinine measurements in men with lower urinary tract symptoms secondary to benign prostatic hyperplasia.
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[Urodynamic study of obstructive uropathy due to benign prostatic hyperplasia].
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Combination of intravesical prostatic protrusion and resistive index is useful to predict bladder outlet obstruction in patients with lower urinary tract symptoms suggestive of benign prostatic hyperplasia.膀胱内前列腺突出与阻力指数相结合有助于预测提示良性前列腺增生的下尿路症状患者的膀胱出口梗阻。
Int J Urol. 2016 Nov;23(11):929-933. doi: 10.1111/iju.13188. Epub 2016 Aug 21.

引用本文的文献

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Effects of pentoxifylline on renal structure after urethral obstruction in rat: A stereological study.己酮可可碱对大鼠尿道梗阻后肾脏结构的影响:一项体视学研究
Cent European J Urol. 2011;64(1):30-3. doi: 10.5173/ceju.2011.01.art6. Epub 2011 Mar 18.
2
Acute kidney injury in the elderly.老年人急性肾损伤。
Clin Geriatr Med. 2009 Aug;25(3):331-58. doi: 10.1016/j.cger.2009.04.001.
3
Management of extreme azotemia from urinary tract obstruction without dialysis. Clinical correlates and kinetic modeling of the recovery of renal function.
无需透析治疗尿路梗阻所致极重度氮质血症。肾功能恢复的临床关联及动力学模型。
Int Urol Nephrol. 2007;39(2):587-93. doi: 10.1007/s11255-006-9035-3. Epub 2007 Feb 21.
4
Medical therapy for benign prostatic hyperplasia progression.良性前列腺增生进展的医学治疗。
Curr Urol Rep. 2002 Aug;3(4):269-75. doi: 10.1007/s11934-002-0047-y.
5
Reduction in renal haemodynamics by exaggerated vesicovascular reflex in rats with acute urinary retention.急性尿潴留大鼠中过度的膀胱血管反射导致肾血流动力学降低。
J Physiol. 2000 Jul 15;526 Pt 2(Pt 2):397-408. doi: 10.1111/j.1469-7793.2000.t01-1-00397.x.