Prostaglandin E2 and leukotrienE C4 modulate interleukin-1 inhibition of WEHI-3B tumor cell growth in vitro.
作者信息
Elliott G R, Tak C, Bonta I L
机构信息
Department of Pharmacology, Erasmus University, Rotterdam, The Netherlands.
出版信息
Agents Actions. 1989 Jan;26(1-2):165-6. doi: 10.1007/BF02126595.
PMID:2469320
Abstract
摘要
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Prostaglandin E2 and leukotrienE C4 modulate interleukin-1 inhibition of WEHI-3B tumor cell growth in vitro.前列腺素E2和白三烯C4在体外调节白细胞介素-1对WEHI-3B肿瘤细胞生长的抑制作用。
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本文引用的文献
1
Immunomodulatory-antiinflammatory functions of E-type prostaglandins. Minireview with emphasis on macrophage-mediated effects.E型前列腺素的免疫调节抗炎功能。重点关注巨噬细胞介导效应的综述
Int J Immunopharmacol. 1982;4(2):103-9. doi: 10.1016/0192-0561(82)90057-1.
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Autoinduction of differentiation in WEHI-3B leukemia cells.WEHI-3B白血病细胞中分化的自诱导
Int J Cancer. 1982 Dec 15;30(6):773-80. doi: 10.1002/ijc.2910300616.
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The biology of interleukin 1 and comparison to tumor necrosis factor.白细胞介素1的生物学特性及其与肿瘤坏死因子的比较。
Immunol Lett. 1987 Dec;16(3-4):227-31. doi: 10.1016/0165-2478(87)90151-9.
4
Prostaglandins as endogenous mediators of interleukin 1 production.前列腺素作为白细胞介素1产生的内源性介质。
J Immunol. 1986 Jan;136(1):186-92.
5
Leukotrienes augment interleukin 1 production by human monocytes.白三烯可增强人单核细胞白细胞介素1的产生。
J Immunol. 1985 Dec;135(6):3958-61.
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