Guo Hangyuan, Xing Yangbo, Xu Fukang
Department of Cardiology, Shaoxing People's Hospital, Shaoxing Hospital of zhejiang University, Shaoxing, China.
Clin Pract. 2011 Dec 5;1(4):e104. doi: 10.4081/cp.2011.e104. eCollection 2011 Sep 28.
A 65-year-old man was admitted as for one month of repetitive dizziness and one episode of syncope. Electrocardiogram showed sinus bradycardia and his Holter monitoring also showed sinus bradycardia with sinus arrest, sino-atrial block and a longest pause of 4.3 s. Then sick sinus syndrome and Adam-Stokes syndrome were diagnosed. Then a dual chamber pacemaker (Medtronic SDR303) was implanted and the parameters were normal by detection. The patient was discharged 1 week later with suture removed. Then 1.5 month late the patient was presented to hospital once again for sudden onset of chest pain with exacerbation after taking deep breath. Pacemaker programming showed both pacing and sensing abnormality with threshold of > 5.0V and resistance of 1200Ω. Lead perforation was revealed by chest X-ray and confirmed by echocardiogram. Considering the fact that there was high risk to remove ventricular lead, spiral tip of previous ventricular lead was withdrew followed by implantation of a new ventricular active lead to the septum. Previous ventricular lead was maintained. As we know that the complications of lead perforation in the clinic was rare. Here we discuss the clinical management and the possible reasons for cardiac perforation of active ventricular lead.
一名65岁男性因反复头晕1个月和1次晕厥入院。心电图显示窦性心动过缓,动态心电图监测也显示窦性心动过缓伴窦性停搏、窦房阻滞,最长停搏4.3秒。随后诊断为病态窦房结综合征和阿-斯综合征。接着植入了双腔起搏器(美敦力SDR303),检测显示参数正常。患者1周后出院,缝线拆除。然后在1.5个月后,患者再次因深呼吸后突发胸痛且加重入院。起搏器程控显示起搏和感知均异常,阈值>5.0V,电阻1200Ω。胸部X线显示导线穿孔,超声心动图证实。考虑到取出心室导线风险高,将先前心室导线的螺旋头撤回,随后在室间隔植入一根新的心室主动导线。先前的心室导线予以保留。我们知道临床上导线穿孔的并发症很少见。在此我们讨论心室主动导线穿孔的临床处理及可能原因。