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Multiple mechanisms of vanadate-induced cell killing.

作者信息

Richelmi P, Berté F, Mirabelli F, Bellomo G

机构信息

Istituto di Farmacologia II, University of Pavia, Italy.

出版信息

Biol Trace Elem Res. 1989 Jul-Sep;21:331-5. doi: 10.1007/BF02917272.

DOI:10.1007/BF02917272
PMID:2484609
Abstract
摘要

相似文献

1
Multiple mechanisms of vanadate-induced cell killing.钒酸盐诱导细胞死亡的多种机制。
Biol Trace Elem Res. 1989 Jul-Sep;21:331-5. doi: 10.1007/BF02917272.
2
Mechanisms of vanadate-induced cellular toxicity: role of cellular glutathione and NADPH.钒酸盐诱导细胞毒性的机制：细胞内谷胱甘肽和烟酰胺腺嘌呤二核苷酸磷酸的作用。
Arch Biochem Biophys. 2002 Oct 1;406(1):65-72. doi: 10.1016/s0003-9861(02)00408-3.
3
Impaired antioxidant status in diabetic rat liver. Effect of vanadate.糖尿病大鼠肝脏抗氧化状态受损。钒酸盐的作用。
Biochem Pharmacol. 1993 Feb 9;45(3):539-42. doi: 10.1016/0006-2952(93)90124-f.
4
Vanadate-induced toxicity towards isolated perfused rat livers: the role of lipid peroxidation.
Toxicology. 1991 Feb 11;66(1):63-74. doi: 10.1016/0300-483x(91)90178-4.
5
Vanadate oligomers: in vivo effects in hepatic vanadium accumulation and stress markers.钒酸盐低聚物：对肝脏钒积累和应激标志物的体内影响
J Inorg Biochem. 2005 May;99(5):1238-44. doi: 10.1016/j.jinorgbio.2005.02.023.
6
Decreased resistance to Listeria monocytogenes in mice following vanadate exposure: effects upon the function of macrophages.钒酸盐暴露后小鼠对单核细胞增生李斯特菌的抵抗力降低：对巨噬细胞功能的影响。
Int J Immunopharmacol. 1989;11(3):285-92. doi: 10.1016/0192-0561(89)90166-5.
7
On the role of mitochondria in cell injury caused by vanadate-induced Ca2+ overload.关于线粒体在钒酸盐诱导的Ca2+过载所致细胞损伤中的作用
Toxicology. 1989 Jul 3;57(1):29-44. doi: 10.1016/0300-483x(89)90032-2.
8
Vanadate induces necrotic death in neonatal rat cardiomyocytes through mitochondrial membrane depolarization.钒酸盐通过线粒体膜去极化诱导新生大鼠心肌细胞坏死性死亡。
Chem Res Toxicol. 2008 Mar;21(3):607-18. doi: 10.1021/tx700204r. Epub 2008 Feb 6.
9
The effect of dietary mercury on vanadate toxicity in the chick.日粮汞对雏鸡钒酸盐毒性的影响。
Biol Trace Elem Res. 1989;23:11-6. doi: 10.1007/BF02917174.
10
Interaction of vanadate and chloride in chicks.
Biol Trace Elem Res. 1989;23:1-10. doi: 10.1007/BF02917173.

本文引用的文献

1
Inhibition of lipid peroxidation without prevention of cellular injury in isolated rat hepatocytes.在离体大鼠肝细胞中抑制脂质过氧化但不预防细胞损伤。
Toxicol Appl Pharmacol. 1981 Mar 30;58(1):8-18. doi: 10.1016/0041-008x(81)90110-1.
2
Regulation of intracellular calcium compartmentation: studies with isolated hepatocytes and t-butyl hydroperoxide.细胞内钙区隔化的调节：对分离的肝细胞和叔丁基过氧化氢的研究
Proc Natl Acad Sci U S A. 1982 Nov;79(22):6842-6. doi: 10.1073/pnas.79.22.6842.
3
Vanadate and its significance in biochemistry and pharmacology.
Biochem Pharmacol. 1984 Apr 1;33(7):945-50. doi: 10.1016/0006-2952(84)90498-2.
4
Glutathione reduces cytoplasmic vanadate. Mechanism and physiological implications.谷胱甘肽可还原细胞质中的钒酸盐。作用机制及生理意义。
Biochim Biophys Acta. 1980 Apr 17;629(1):95-106. doi: 10.1016/0304-4165(80)90268-8.
5
Calcium transport and phosphorylated intermediate of (Ca2+ + Mg2+)-ATPase in plasma membranes of rat liver.大鼠肝细胞膜中（Ca2+ + Mg2+）-ATP酶的钙转运及磷酸化中间体
J Biol Chem. 1983 Apr 10;258(7):4404-10.
6
Cytotoxicity-related alterations of selected cellular functions after in vitro vanadate exposure.体外钒酸盐暴露后所选细胞功能的细胞毒性相关改变。
Biochem Pharmacol. 1985 Jul 15;34(14):2465-70. doi: 10.1016/0006-2952(85)90527-1.
7
The regulation of intracellular calcium by mitochondria.线粒体对细胞内钙的调节。
Ann N Y Acad Sci. 1978 Apr 28;307:269-84. doi: 10.1111/j.1749-6632.1978.tb41957.x.