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HLA-B*51 the primary risk in Behçet disease.

作者信息

Wallace Graham R

机构信息

Centre for Translational Inflammation Research, University of Birmingham, Birmingham West Midlands B15 2TT, United Kingdom

出版信息

Proc Natl Acad Sci U S A. 2014 Jun 17;111(24):8706-7. doi: 10.1073/pnas.1407307111. Epub 2014 May 29.

DOI:10.1073/pnas.1407307111

PMID:24876276

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4066492/

Abstract

摘要

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Critical role of endoplasmic reticulum aminopeptidase 1 in determining the length and sequence of peptides bound and presented by HLA-B27.

Arthritis Rheumatol. 2014 Feb;66(2):284-94. doi: 10.1002/art.38249.

Behçet's syndrome.

Rheum Dis Clin North Am. 2013 May;39(2):245-61. doi: 10.1016/j.rdc.2013.02.010. Epub 2013 Mar 14.

Identification of multiple independent susceptibility loci in the HLA region in Behçet's disease.

Nat Genet. 2013 Mar;45(3):319-24. doi: 10.1038/ng.2551. Epub 2013 Feb 10.

Genome-wide association analysis identifies new susceptibility loci for Behçet's disease and epistasis between HLA-B*51 and ERAP1.

Nat Genet. 2013 Feb;45(2):202-7. doi: 10.1038/ng.2520. Epub 2013 Jan 6.

Genome-wide association study identifies GIMAP as a novel susceptibility locus for Behcet's disease.

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An NKG2D-mediated human lymphoid stress surveillance response with high interindividual variation.

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HLA-B*51 the primary risk in Behçet disease.

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