Department of Neurology, Mainz University, Mainz, Germany.
Eur J Neurol. 2015 Feb;22(2):416-8. doi: 10.1111/ene.12475. Epub 2014 Jun 19.
There is little evidence about the site where compensatory vestibular mechanisms in patients with cerebellar strokes take place.
To determine whether the location of a cerebellar lesion might be a crucial variable in vestibular compensation a sample of 22 patients with cerebellar stroke were tested for graviceptive function in the acute and chronic stage.
Our statistical anatomical lesion analysis indicated that mainly lesions of the cerebellar hemispheres (lobule V, VI, VIIa) hinder vestibular compensation and might lead to an overcompensation.
Overcompensation-induced dysfunction can be explained by the absence of cerebellar inhibitory signals.
关于小脑卒中患者代偿性前庭机制发生的部位，相关证据很少。
为了确定小脑病变的位置是否是前庭代偿的关键变量，我们对 22 例小脑卒中患者的前庭功能进行了急性和慢性阶段的测试。
我们的统计解剖病变分析表明，主要是小脑半球（小叶 V、VI、VIIa）的病变阻碍了前庭代偿，并可能导致过度代偿。
小脑抑制信号缺失可以解释过度代偿引起的功能障碍。
