类风湿关节炎：类风湿关节炎中的应激：促炎途径的触发因素？ - Suppr

Rheumatoid arthritis: Stress in RA: a trigger of proinflammatory pathways?

作者信息

Straub Rainer H

机构信息

Laboratory of Experimental Rheumatology and Neuroendocrino-Immunology, Division of Rheumatology, Department of Internal Medicine I, University Hospital, Regensburg 93042, Germany.

出版信息

Nat Rev Rheumatol. 2014 Sep;10(9):516-8. doi: 10.1038/nrrheum.2014.110. Epub 2014 Jul 8.

DOI:10.1038/nrrheum.2014.110

PMID:25003764

Abstract

摘要

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Rheumatoid arthritis: Stress in RA: a trigger of proinflammatory pathways?类风湿关节炎：类风湿关节炎中的应激：促炎途径的触发因素？

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本文引用的文献

Immune responses to stress in rheumatoid arthritis and psoriasis.类风湿关节炎和银屑病的应激免疫反应。

Rheumatology (Oxford). 2014 Oct;53(10):1844-8. doi: 10.1093/rheumatology/keu221. Epub 2014 May 20.

Assessment of plasma C-reactive protein as a biomarker of posttraumatic stress disorder risk.评估血浆 C 反应蛋白作为创伤后应激障碍风险的生物标志物。

JAMA Psychiatry. 2014 Apr;71(4):423-31. doi: 10.1001/jamapsychiatry.2013.4374.

Genetics of rheumatoid arthritis contributes to biology and drug discovery.类风湿关节炎的遗传学研究有助于生物学和药物发现。

Nature. 2014 Feb 20;506(7488):376-81. doi: 10.1038/nature12873. Epub 2013 Dec 25.

Energy regulation and neuroendocrine-immune control in chronic inflammatory diseases.慢性炎症性疾病中的能量调节与神经内分泌-免疫控制。

J Intern Med. 2010 Jun;267(6):543-60. doi: 10.1111/j.1365-2796.2010.02218.x. Epub 2010 Jan 28.

The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.泛素编辑酶A20（TNFAIP3）是免疫病理学的核心调节因子。

Trends Immunol. 2009 Aug;30(8):383-91. doi: 10.1016/j.it.2009.05.007. Epub 2009 Jul 28.

Stress of different types increases the proinflammatory load in rheumatoid arthritis.不同类型的压力会增加类风湿关节炎的促炎负荷。

Arthritis Res Ther. 2009;11(3):114. doi: 10.1186/ar2712. Epub 2009 Jun 17.

The effects of acute psychological stress on circulating inflammatory factors in humans: a review and meta-analysis.急性心理应激对人体循环炎症因子的影响：一项综述与荟萃分析。

Brain Behav Immun. 2007 Oct;21(7):901-12. doi: 10.1016/j.bbi.2007.03.011. Epub 2007 May 1.

Stress-induced immune dysfunction: implications for health.应激诱导的免疫功能障碍：对健康的影响。

Nat Rev Immunol. 2005 Mar;5(3):243-51. doi: 10.1038/nri1571.

A mechanism converting psychosocial stress into mononuclear cell activation.一种将心理社会压力转化为单核细胞激活的机制。

Proc Natl Acad Sci U S A. 2003 Feb 18;100(4):1920-5. doi: 10.1073/pnas.0438019100. Epub 2003 Feb 10.

Anti-inflammatory cooperativity of corticosteroids and norepinephrine in rheumatoid arthritis synovial tissue in vivo and in vitro.皮质类固醇与去甲肾上腺素在类风湿性关节炎滑膜组织中的体内外抗炎协同作用

FASEB J. 2002 Jul;16(9):993-1000. doi: 10.1096/fj.02-0085com.

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Rheumatoid arthritis: Stress in RA: a trigger of proinflammatory pathways?

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