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肠致病性大肠杆菌map在体内的表达与其他III型分泌效应蛋白的表达显著不同。

Expression of enteropathogenic Escherichia coli map is significantly different than that of other type III secreted effectors in vivo.

作者信息

Nguyen Mai, Rizvi Jason, Hecht Gail

机构信息

Departments of Medicine, Microbiology/Immunology, Division of Gastroenterology, Loyola University Chicago, Stritch School of Medicine, Maywood, Illinois, USA.

Jefferson Medical College, Philadelphia, Pennsylvania, USA.

出版信息

Infect Immun. 2015 Jan;83(1):130-7. doi: 10.1128/IAI.02467-14. Epub 2014 Oct 13.

Abstract

The enteropathogenic Escherichia coli (EPEC) locus of enterocyte effacement (LEE)-encoded effectors EspF and Map are multifunctional and have an impact on the tight junction barrier while the non-LEE-encoded proteins NleH1 and NleH2 possess significant anti-inflammatory activity. In order to address the temporal expression of these important genes in vivo, their promoters were cloned upstream of the luxCDABE operon, and luciferase expression was measured in EPEC-infected mice by bioluminescence using an in vivo imaging system (IVIS). Bioluminescent images of living mice, of excised whole intestines, and of whole intestines longitudinally opened and washed were assessed. The majority of bioluminescent bacteria localized in the cecum by 3 h postinfection, indicating that the cecum is not only a major colonization site of EPEC but also a site of EPEC effector gene expression in mice. espF, nleH1, and nleH2 were abundantly expressed over the course of infection. In contrast, map expression was suppressed at 2 days postinfection, and at 4 days postinfection it was totally abolished. After 2 to 4 days postinfection, when map is suppressed, EPEC colonization is significantly reduced, indicating that map may be one of the factors required to maintain EPEC colonization. This was confirmed in a competitive colonization study and in two models of chronic infection, repeated exposure to ketamine and Citrobacter rodentium infection. Our data suggest that map expression contributes to the maintenance of EPEC colonization.

摘要

肠道致病性大肠杆菌(EPEC)的肠细胞脱落位点(LEE)编码的效应蛋白EspF和Map具有多种功能,会影响紧密连接屏障,而非LEE编码的蛋白NleH1和NleH2具有显著的抗炎活性。为了研究这些重要基因在体内的表达时间,将它们的启动子克隆到luxCDABE操纵子的上游,并通过体内成像系统(IVIS)利用生物发光法在感染EPEC的小鼠中测量荧光素酶的表达。评估了活小鼠、切除的整个肠道以及纵向切开并冲洗后的整个肠道的生物发光图像。感染后3小时,大多数生物发光细菌定位于盲肠,这表明盲肠不仅是EPEC的主要定植部位,也是EPEC效应基因在小鼠中表达的部位。在感染过程中,espF、nleH1和nleH2大量表达。相比之下,map的表达在感染后2天受到抑制,在感染后4天则完全消失。在感染后2至4天,当map受到抑制时,EPEC的定植显著减少,这表明map可能是维持EPEC定植所需的因素之一。这在竞争性定植研究以及两种慢性感染模型(反复接触氯胺酮和啮齿柠檬酸杆菌感染)中得到了证实。我们的数据表明map的表达有助于维持EPEC的定植。

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