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Brown adipose tissue and the cold war against obesity.

作者信息

Calvani Riccardo, Leeuwenburgh Christiaan, Marzetti Emanuele

机构信息

Department of Geriatrics, Neurosciences and Orthopedics, Catholic University of the Sacred Heart, Rome, Italy.

Department of Aging and Geriatric Research, University of Florida, Gainesville, FL

出版信息

Diabetes. 2014 Dec;63(12):3998-4000. doi: 10.2337/db14-1373.

DOI:10.2337/db14-1373
PMID:25414014
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4392919/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/5e16/4392919/2f517077f608/3998fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/5e16/4392919/2f517077f608/3998fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/5e16/4392919/2f517077f608/3998fig1.jpg

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Cell Metab. 2014 Sep 2;20(3):396-407. doi: 10.1016/j.cmet.2014.07.005. Epub 2014 Aug 7.
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Surgery for weight loss in adults.成人减肥手术。
D-甘露醇通过β3-肾上腺素能受体依赖性机制诱导棕色脂肪样表型。
Cells. 2021 Mar 31;10(4):768. doi: 10.3390/cells10040768.
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A Peripheral CB1R Antagonist Increases Lipolysis, Oxygen Consumption Rate, and Markers of Beiging in 3T3-L1 Adipocytes Similar to RIM, Suggesting that Central Effects Can Be Avoided.外周 CB1R 拮抗剂增加 3T3-L1 脂肪细胞的脂肪分解、耗氧率和米色标记物，类似于 RIM，表明可以避免中枢作用。
Int J Mol Sci. 2020 Sep 10;21(18):6639. doi: 10.3390/ijms21186639.
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Activation of NPRs and UCP1-independent pathway following CB1R antagonist treatment is associated with adipose tissue beiging in fat-fed male dogs.大麻素 1 型受体拮抗剂治疗后激活 NPRs 和 UCP1 非依赖性途径与肥胖雄性犬脂肪组织米色化有关。
Am J Physiol Endocrinol Metab. 2019 Sep 1;317(3):E535-E547. doi: 10.1152/ajpendo.00539.2018. Epub 2019 Jun 25.
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BJR Case Rep. 2015 Jul 29;1(3):20150204. doi: 10.1259/bjrcr.20150204. eCollection 2015.
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